Is proviron not simply straight DHT? How is that any different than the DHT used in the studies ur referencing?Men with low serum estradiol levels have low tissue, estradiol levels, because what you measure in the serum is actually spill over from the tissues where it is made. When you lower body fat, you also lower estradiol. So when you take anabolic steroids, have any kind, you're going to have an increase in lean muscle mass and decrease in fat mass, which will decrease estradiol. But what happens when you take that aromatase inhibitor or lower your estradiol is that it increases your visceral body fat. That's why bodybuilders die from the inside out it is from the aromatase inhibition. Bodybuilders have low estradiol levels, cause they have not much adipose tissue subcutaneously, which therefore will not convert a lot of the testosterone to estradiol that you're measuring in the serum. That's where most of it comes from from the peripheral aromatization and more specifically the adipose tissue. But look man what you're talking about when it comes to the anabolic steroids have absolutely nothing and I mean nothing to do with what I do. It has no place in testosterone therapy or hormone replacement therapy at all. Your questions do not relate to people that are on hormone replacement therapy or testosterone therapy. You're a proper to the problem not the solution. What I mean by that is anabolic steroid use, and the use of all these mixtures of anabolic substances has zero to do with hormone replacement therapy in normal men and women. It is where all of the misinformation comes from as well.
In healthy men , serum levels of E2 reflect the total E2 that has diffused into the blood from all tissues having been synthesized by aromatase and escaped local tissue metabolism. These blood levels, no matter how accurately measured, are an indirect reflection of estrogen at the tissue level
testosterone and nandrolone are both anabolic steroids, but they’re used and talked about, in regards to HRT. How is primobolan any different than an AAS that’s commonly used in HRT?
u have no answer because the fact that a DHT derivative like primobolan lowers E2 in the serum, and masteron and Proviron inhibit estrogen on a tissue/ cell level, destroys ur theory that DHT has no estrogen inhibiting effects, whether it’s at the aromatase level, or at the tissue/ cell/ receptor level. Ur stuck on one theory, and won’t even open ur mind to the possibility that DHT can affect estrogen in other ways. I would say people that are close minded and stuck on one theory about how something works is the actual problem in this community. I do like ur ideas and thoughts and opinions on the majority of things tho, so I do appreciate and enjoy ur posts. But I’m just not sure if DHT has zero affects on estrogen, and it would be nice if u were more open to brainstorming about whether it does or not, rather than being stuck on studies done on males that aren’t on exogenous androgens. That’s not this community, and it doesn’t really apply to guys on HRT. So for u to extrapolate a theory from studies done on natural males, and apply it to males on exogenous testosterone, and be so confident about this theory, seems very short sighted and lacking critical thinking skills, imo