There's this complexity, and probably even more. I have dabbled with anastrozole when my TRT dose was higher, and I also had a period with very low estradiol for some other reason. I don't recall that these had much effect on motivation. The anastrozole would dampen excessive emotionalism. Very low estradiol totally knocked out libido and made me feel emotionally flat.
My E2 was 57 pg/mL when TT was close to 1,250 ng/dL. Interestingly that's one of my lower aromatization rates when not on an AI. I wonder if the saturation effect was starting to kick in? I also see that early on I was using Androgel and measured TT ~600 ng/dL and E2 ~20 pg/mL, an even lower rate of aromatization, perhaps due to the DHT.
I doubt body fat is much of an issue. Not that it can be trusted, but the scale puts BF at 12%. In any case, I would claim to be skinny-muscular, not skinny-fat. Similarly I doubt insulin is much of an issue. Last year the fasting value was 2 uIU/mL, below the reference range. I expect my skepticism would survive the experience of 1,200 ng/dL TT and 30 pg/mL E2. The enhanced athleticism would again be fun. But if the headaches returned then stopping would be a no-brainer, pardon the pun. Similarly, the deteriorating lipids would not be appreciated. With respect to libido and sexual function, I have to mention again that even with that recent excessive E2/T ratio and low TT, these parameters were more consistently good than at any other time on TRT. Admittedly there's added complexity here with my continued use of kisspeptin-10 and gonadorelin. But my expectation is that knocking these out would simply make things worse. I assume your suggested experiment results in full HPTA suppression? I also assume that hCG use would be excluded because that would make it virtually impossible to keep E2 down at 30 pg/mL naturally.
Though Vince doesn't seem to get it, I think I've made it pretty clear that I'm not opposed to informed individuals such as yourself experimenting with TRT doses and ancillaries as you see fit. What bothers me is seeing this parade of guys who are being harmed by high doses given right from the start. As I have said many times before, these situations often get worse when the symptoms of excess are not addressed by lowering the dose. Instead we see bloodletting for high HCT and AIs for high E2. More subtle issues often remain unaddressed. I believe that some fraction of men will have problems from the secondary effects of HPTA shutdown. But at this time there's little official recognition of this, or of the possibility that short-acting testosterone is a solution.