Anyone been on 250 IU HCG with TC and not needed an AI?

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Westin

Active Member
I’ve been doing 375 IU in addition to 120mg TC split in 2, and have had to use .25 of an AI. I just dropped to 250 IU 3 X weekly. Wondering if even at 250 IU my E2 get to where I feel the high E2 symptoms.

Anyone out on 250 iu without an AI?
 
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Defy Medical TRT clinic doctor
I take 400 IU HCG 3 x weekly along with 210 mg TC and only need 0.125 mg anastrozole 1x weekly. DIM/Calcium d-glucarate significantly helps me manage estrogen.

This is the supplement I take that contains DIM/Calcium d-glucarate. Robot Check

It's worth a shot.
 
I take 400 IU HCG 3 x weekly along with 210 mg TC and only need 0.125 mg anastrozole 1x weekly. DIM/Calcium d-glucarate significantly helps me manage estrogen.

This is the supplement I take that contains DIM/Calcium d-glucarate. Robot Check

It's worth a shot.

What were your E2 #’s prior to the AI or did you start the AI in the beginning? And current #’s?

Thanks much.
 
I do 250 iu of HcG twice a week and on TRT. No AI for a while now. My TRT dose is now only 84mg/week. I do 0.2ml twice a week now. My vial is a 210mg/ml. 10 ml. Before I reduced my test I was doing approx 120-130mg of test per week no AI. Here were my last results from March 5.
04F0C4E4-0A32-4F98-8793-844840078A8C.jpeg
 
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I do 250 iu of HcG twice a week and on TRT. No AI for a while now. My TRT dose is now only 84mg/week. I do 0.2ml twice a week now. My vial is a 210mg/ml. 10 ml. Before I reduced my test I was doing approx 120-130mg of test per week no AI. Here were my last results from March 5. View attachment 9460

Were you doing that with HCG and no AI?
 
If I Just take my 150 mg per week T, and only ONCE per week HCG of 300 iu I do not need an AI. Anything above that once per week 300 iu, means I need an AI.
 
I tried different dosing and frequency of HCG with my T and didnt need an AI.
For my body 150IU HCG with my T, ED and SubQ works best, no need for an AI. I do use Zinc ad a DHT derivative which are known to inhibit estrogen aromatisation and receptor binding.
 
I tried different dosing and frequency of HCG with my T and didnt need an AI.
For my body 150IU HCG with my T, ED and SubQ works best, no need for an AI. I do use Zinc ad a DHT derivative which are known to inhibit estrogen aromatisation and receptor binding.
Proviron?
 
What were your E2 #’s prior to the AI or did you start the AI in the beginning? And current #’s?

Thanks much.
I don’t have blood work from before. I’ve been on Test for 10 years and TRT for 5 years. I didn’t start an AI until about 4 years ago, but I don’t have readily available bloodwork. I keep my E2 between 50-70 pg/dL and my TT between 1000-1200 ng/dL.
 
I don’t have blood work from before. I’ve been on Test for 10 years and TRT for 5 years. I didn’t start an AI until about 4 years ago, but I don’t have readily available bloodwork. I keep my E2 between 50-70 pg/dL and my TT between 1000-1200 ng/dL.

Helpful. Thanks.
 
Im on 150iu daily with 10mgprop daily. interstingly a few days ago i mssed hcg dose and following day i injected 250iu and libido went crazy about 6 hours later and lasted about a day.

this guy believes that the primary libido function comes from estrogen and maintains libido with topical estrogen while he experiments with a few sarms and also Nandrolone for trt in various experiments with extensive bloodwork. Point is he still has libido with no testosterone and therefore no dht with these other compounds taking care of anabolism and maintaining his size and physic but avoiding androgenic effects.

Hes not saying that its preferable but he is saying it has its posible and may have benefits and that most of the point of testosterone is to just maintain a physiological amount of estrogen.

edit: hes also low shbg under 20 always and also says he excretes estrogen quickly. hes also used dht blockers with no ill effect. also he has a history of AAS use.

 
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Im on 150iu daily with 10mgprop daily. interstingly a few days ago i mssed hcg dose and following day i injected 250iu and libido went crazy about 6 hours later and lasted about a day.

this guy points out that the primary libido function comes from estrogen and maintains libido with topical estrogen while he experiments with a few sarms and also Nandrolone for trt in various experiments with extensive bloodwork. Point is he still has libido with no testosterone and therefore no dht with these other compounds taking care of anabolism and maintaining his size and physic but avoiding androgenic effects.

Hes not saying that its preferable but he is saying it has its posible and may have benefits and that most of the point of testosterone is to just maintain a physiological amount of estrogen.

edit: hes also low shbg under 20 always and also says he excretes estrogen quickly. hes also used dht blockers with no ill effect. also he has a history of AAS use.

Just for clarification, the fellow posits a hypothesis that primary libido comes from estrogen. This is not actually shown in studies to be true, and thus should not be touted as fact.

Testosterone is necessary for myriad functions, not least of which is its role in erectile function stemming from its conversion into DHT. Nitric-oxide mediated erectile function can only occur in the presence of adequate DHT, thus necessitating the need for adequate amounts of DHT for erectile function. Research-based evidence discusses this, and I can share if you would like to see.

DHT is also vital for cognitive performance, the lack of which leads to a decline in cognitive function.

These are just 2 of myriad functions that Testosterone plays a vital role in. Bro knowledge is more often than not misleading and based on logical fallacies and unproven hypotheses.

I do want to make clear that I’m not suggesting that estrogen plays no role in libido, because it certainly does play a vital role. Estrogen, however, is not the primary nor the sole component in libido or erectile function.
 
Just for clarification, the fellow posits a hypothesis that primary libido comes from estrogen. This is not actually shown in studies to be true, and thus should not be touted as fact.

Testosterone is necessary for myriad functions, not least of which is its role in erectile function stemming from its conversion into DHT. Nitric-oxide mediated erectile function can only occur in the presence of adequate DHT, thus necessitating the need for adequate amounts of DHT for erectile function. Research-based evidence discusses this, and I can share if you would like to see.

DHT is also vital for cognitive performance, the lack of which leads to a decline in cognitive function.

These are just 2 of myriad functions that Testosterone plays a vital role in. Bro knowledge is more often than not misleading and based on logical fallacies and unproven hypotheses.

I do want to make clear that I’m not suggesting that estrogen plays no role in libido, because it certainly does play a vital role. Estrogen, however, is not the primary nor the sole component in libido or erectile function.

i couldnt agree more with the physiological relevance of testosterone and its implications in the male metabolism. Tho I did find it interesting his personal trial with the sarm LGD and estrogen claiming his libido was near normal. There would be no DHT in this scenario. I do not know other factors or ill effects but he is usually pretty objective when it comes to describing pros cons of his little escapades. Hes actually got a few though provoking takes on things.
 
I don’t do hypothesis, I do fact. You can’t have one (test) without the other (E2). It’s a balance. One without the other = 0 results.
 
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