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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
Effect of TRT vs hCG/FSH on upstream hormone pathways.
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<blockquote data-quote="madman" data-source="post: 186175" data-attributes="member: 13851"><p><strong>Δ4 pathway </strong></p><p><strong>Progesterone, 17-OH-progesterone (17 OHP) </strong></p><p></p><p><span style="color: rgb(44, 130, 201)"><strong><u><em>One major finding of this study is that hCG/rFSH replacement stimulates and thus normalizes some important steroid hormones belonging to the Δ4 pathway of steroidogenesis in hypogonadotropic hypogonadal males</em></u><em>.</em></strong></span><em> <span style="color: rgb(184, 49, 47)"><strong><u>17-OHP levels were significantly decreased on treatment with testosterone, but not while the males were on gonadotropin replacement, indicating that a major proportion of this precursor steroid is produced in the gonads and requires gonadotropin stimulation for secretion, while a minor part of it stems from other sources</u>.</strong></span> It is likely that ACTH- stimulated production in the adrenal gland contributes to it.</em></p><p></p><p></p><p></p><p>Was going to post yesterday but forgot.</p><p></p><p>A pure joy to read.</p><p></p><p>Thanks to one of the brightest minds who has contributed greatly to the field of endocrinology and andrology.</p><p></p><p><strong>Julia Rohayem , Michael Zitzmann , Sandra Laurentino , Sabine Kliesch , <span style="color: rgb(184, 49, 47)">Eberhard Nieschlag</span>, Paul Martin Holterhus, Alexandra Kulle</strong></p><p><strong></strong></p><p><strong></strong></p><p><strong></strong></p><p><strong></strong></p><p><strong></strong></p><p><strong></strong></p><p><strong>Limitations </strong></p><p><strong></strong></p><p><strong><span style="color: rgb(184, 49, 47)"><em>One limitation of the present study, regarding the investigation of gonadotropin-effects is that hCG was used to replace LH.</em></span></strong> <strong><em><span style="color: rgb(44, 130, 201)"><u>T</u></span></em><span style="color: rgb(44, 130, 201)"><u><strong><em>h</em></strong></u><strong><em><u>is substance may have slightly different properties regarding the activation of LHCG receptors</u> 55. However, at present, rLH is not licensed for clinical use in males. </em></strong></span></strong></p></blockquote><p></p>
[QUOTE="madman, post: 186175, member: 13851"] [B]Δ4 pathway Progesterone, 17-OH-progesterone (17 OHP) [/B] [COLOR=rgb(44, 130, 201)][B][U][I]One major finding of this study is that hCG/rFSH replacement stimulates and thus normalizes some important steroid hormones belonging to the Δ4 pathway of steroidogenesis in hypogonadotropic hypogonadal males[/I][/U][I].[/I][/B][/COLOR][I] [COLOR=rgb(184, 49, 47)][B][U]17-OHP levels were significantly decreased on treatment with testosterone, but not while the males were on gonadotropin replacement, indicating that a major proportion of this precursor steroid is produced in the gonads and requires gonadotropin stimulation for secretion, while a minor part of it stems from other sources[/U].[/B][/COLOR] It is likely that ACTH- stimulated production in the adrenal gland contributes to it.[/I] Was going to post yesterday but forgot. A pure joy to read. Thanks to one of the brightest minds who has contributed greatly to the field of endocrinology and andrology. [B]Julia Rohayem , Michael Zitzmann , Sandra Laurentino , Sabine Kliesch , [COLOR=rgb(184, 49, 47)]Eberhard Nieschlag[/COLOR], Paul Martin Holterhus, Alexandra Kulle Limitations [COLOR=rgb(184, 49, 47)][I]One limitation of the present study, regarding the investigation of gonadotropin-effects is that hCG was used to replace LH.[/I][/COLOR][/B] [B][I][COLOR=rgb(44, 130, 201)][U]T[/U][/COLOR][/I][COLOR=rgb(44, 130, 201)][U][B][I]h[/I][/B][/U][B][I][U]is substance may have slightly different properties regarding the activation of LHCG receptors[/U] 55. However, at present, rLH is not licensed for clinical use in males. [/I][/B][/COLOR][/B] [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
Effect of TRT vs hCG/FSH on upstream hormone pathways.
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