Cardic Hypertrophy ventricular

[Tommy Wall]

I made ECG, stress test and Dopple ultrasound. I when I handed in the results to the secretary I was informed that if something is wrong they would call me otherwise I could pick the results up in 5 days. No one called and I picked the results up. I was a bit curious so I read everthing that the doc that made the doppler had written. Everything was normal but there was a line that made me worried. Slight /light cardiac hyperthrophy ventricular. But my GP didn´t say anything about it. My GP knows I´m working out and I don´t know if that can cause this. Shall I worry?

 

[Tommy Wall]

Any input on this?

 

[Vince]

Luckily it's only slight. I believe is caused by high blood pressure and of course being overweight contributes.

 

[madman]

I made ECG, stress test and Dopple ultrasound. I when I handed in the results to the secretary I was informed that if something is wrong they would call me otherwise I could pick the results up in 5 days. No one called and I picked the results up. I was a bit curious so I read everthing that the doc that made the doppler had written. Everything was normal but there was a line that made me worried. Slight /light cardiac hyperthrophy ventricular. But my GP didn´t say anything about it. My GP knows I´m working out and I don´t know if that can cause this. Shall I worry?

https://onlinelibrary.wiley.com/doi/full/10.1111/jch.12977

Conclusions

Even though both hypertensive patients and athletes can develop LVH, there are several structural and physiological differences between the two. That the LV hypertrophies are a function of chronically or intermittently elevated BP is suggested by the aforementioned findings, which show that both fit hypertensive patients and relatively fit individuals are not likely to achieve systolic BP ≥150 mm Hg necessary to stimulate cardiac remodeling during normal daily activities and, therefore, an increase in LVM is not likely. For those with existing LVH, regularly performed aerobic exercise of moderate intensity improves fitness and lowers BP at absolute workloads and the daily hemodynamic load, as is reflected by lower BP. Consequently, the daily exposure to a substantially lower hemodynamic load removes the impetus for cardiac remodeling and eventually leads to LVM regression.