ExcelMale
Menu
Home
What's new
Latest activity
Forums
New posts
Search forums
What's new
New posts
Latest activity
Videos
Lab Tests
Doctor Finder
Buy Books
About Us
Men’s Health Coaching
Log in
Register
What's new
Search
Search
Search titles only
By:
New posts
Search forums
Menu
Log in
Register
Navigation
Install the app
Install
More options
Contact us
Close Menu
Forums
Testosterone Replacement, Low T, HCG, & Beyond
Testosterone and Men's Health Articles
Androgen Treatment in Adolescent Males With Hypogonadism
JavaScript is disabled. For a better experience, please enable JavaScript in your browser before proceeding.
You are using an out of date browser. It may not display this or other websites correctly.
You should upgrade or use an
alternative browser
.
Reply to thread
Message
<blockquote data-quote="madman" data-source="post: 179116" data-attributes="member: 13851"><p>[ATTACH=full]9791[/ATTACH]</p><p><strong><span style="color: rgb(184, 49, 47)">Figure 2.</span> Clinical presentation of male hypogonadism according to the period of life when it is established. The testes differentiate in the first trimester of fetal life, independently of LH and FSH, the pituitary gonadotropins. Testicular hormones drive the internal and external genitalia through the male differentiation pathway. Hypogonadism established in early fetal life leads to the existence of ambiguous or female genitalia in XY newborns. In later fetal life, androgens induce testicular descent and has a trophic effect on the external genitalia. Primary and central hypogonadisms established in this period result in a male newborn with micropenis, micro-orchidism, and/or cryptorchidism. During childhood, gonadotropins and androgens are very low or undetectable; thus, hypoandrogenism is physiological. At pubertal age, the gonadal axis is reactivated, and secondary sex characteristics develop. Hypogonadism may result in absent or incomplete pubertal development, infertility, and/or sexual dysfunction. Modified, with permission, from Grinspon et al. (2019). © 2019 Elsevier Limited. This figure was created using BioRender (<a href="https://biorender.com/" target="_blank">Home</a>). </strong></p></blockquote><p></p>
[QUOTE="madman, post: 179116, member: 13851"] [ATTACH type="full"]9791[/ATTACH] [B][COLOR=rgb(184, 49, 47)]Figure 2.[/COLOR] Clinical presentation of male hypogonadism according to the period of life when it is established. The testes differentiate in the first trimester of fetal life, independently of LH and FSH, the pituitary gonadotropins. Testicular hormones drive the internal and external genitalia through the male differentiation pathway. Hypogonadism established in early fetal life leads to the existence of ambiguous or female genitalia in XY newborns. In later fetal life, androgens induce testicular descent and has a trophic effect on the external genitalia. Primary and central hypogonadisms established in this period result in a male newborn with micropenis, micro-orchidism, and/or cryptorchidism. During childhood, gonadotropins and androgens are very low or undetectable; thus, hypoandrogenism is physiological. At pubertal age, the gonadal axis is reactivated, and secondary sex characteristics develop. Hypogonadism may result in absent or incomplete pubertal development, infertility, and/or sexual dysfunction. Modified, with permission, from Grinspon et al. (2019). © 2019 Elsevier Limited. This figure was created using BioRender ([URL="https://biorender.com/"]Home[/URL]). [/B] [/QUOTE]
Insert quotes…
Verification
Post reply
Share this page
Facebook
Twitter
Reddit
Pinterest
Tumblr
WhatsApp
Email
Share
Link
Sponsors
Forums
Testosterone Replacement, Low T, HCG, & Beyond
Testosterone and Men's Health Articles
Androgen Treatment in Adolescent Males With Hypogonadism
This site uses cookies to help personalise content, tailor your experience and to keep you logged in if you register.
By continuing to use this site, you are consenting to our use of cookies.
Accept
Learn more…
Top