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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone and Men's Health Articles
Androgen Treatment in Adolescent Males With Hypogonadism
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<blockquote data-quote="madman" data-source="post: 179115" data-attributes="member: 13851"><p>[ATTACH=full]9790[/ATTACH]</p><p><strong><span style="color: rgb(184, 49, 47)">Figure 1.</span> Changes in the anatomical aspects and circulating levels of sex hormones in males from fetal life through adulthood. In early fetal life, testicular hormones, which are produced independently of fetal LH and FSH, drive masculinization of the genitalia. In the second part of intrauterine life, luteinizing hormone (LH) and follicle-stimulating hormone (FSH) are the main regulators of gonadal endocrine activity resulting in testicular growth and descent and in enlargement of the external genitalia. During the first 3–6 months after birth (also called “mini-puberty”), gonadotropin and testicular endocrine function remains active. During the rest of infancy and childhood, gonadotropins, testosterone (T), and insulin-like factor 3 (INSL3) decline, but Sertoli cell secretion of anti-Müllerian hormone (AMH) and inhibin B persist at high levels. During pubertal development, the size of the testes increases notoriously due to spermatogenesis, driven by gonadotropins and testosterone. AMH is inhibited by testosterone, while inhibin B secretion is boosted by the action of FSH and germ cells. Reprinted, with permission, from Salonia et al. (2019). ©2019 Springer Nature Limited. </strong></p></blockquote><p></p>
[QUOTE="madman, post: 179115, member: 13851"] [ATTACH type="full"]9790[/ATTACH] [B][COLOR=rgb(184, 49, 47)]Figure 1.[/COLOR] Changes in the anatomical aspects and circulating levels of sex hormones in males from fetal life through adulthood. In early fetal life, testicular hormones, which are produced independently of fetal LH and FSH, drive masculinization of the genitalia. In the second part of intrauterine life, luteinizing hormone (LH) and follicle-stimulating hormone (FSH) are the main regulators of gonadal endocrine activity resulting in testicular growth and descent and in enlargement of the external genitalia. During the first 3–6 months after birth (also called “mini-puberty”), gonadotropin and testicular endocrine function remains active. During the rest of infancy and childhood, gonadotropins, testosterone (T), and insulin-like factor 3 (INSL3) decline, but Sertoli cell secretion of anti-Müllerian hormone (AMH) and inhibin B persist at high levels. During pubertal development, the size of the testes increases notoriously due to spermatogenesis, driven by gonadotropins and testosterone. AMH is inhibited by testosterone, while inhibin B secretion is boosted by the action of FSH and germ cells. Reprinted, with permission, from Salonia et al. (2019). ©2019 Springer Nature Limited. [/B] [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone and Men's Health Articles
Androgen Treatment in Adolescent Males With Hypogonadism
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