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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
18 Year-Old With Testicular Atrophy
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<blockquote data-quote="madman" data-source="post: 137834" data-attributes="member: 13851"><p>LH stimulates the leydig cells in the testes to produce testosterone and FSH stimulates the sertoli/germ cells to produce sperm.</p><p></p><p>When one uses exogenous testosterone/AAS it results in shutdown of the hpta and the pituitary no longer secretes LH/FSH and the leydig cells/seminiferous tubules atrophy.</p><p></p><p>The leydig cells only make up 10-20% of testicular volume as oppose to the seminiferous tubules (where sperm is produced) make up almost 80% so a majority of the shrinkage results from atrophy of the seminiferous tubules.</p></blockquote><p></p>
[QUOTE="madman, post: 137834, member: 13851"] LH stimulates the leydig cells in the testes to produce testosterone and FSH stimulates the sertoli/germ cells to produce sperm. When one uses exogenous testosterone/AAS it results in shutdown of the hpta and the pituitary no longer secretes LH/FSH and the leydig cells/seminiferous tubules atrophy. The leydig cells only make up 10-20% of testicular volume as oppose to the seminiferous tubules (where sperm is produced) make up almost 80% so a majority of the shrinkage results from atrophy of the seminiferous tubules. [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
18 Year-Old With Testicular Atrophy
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