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Thyroid, Pregnenolone, Progesterone, DHEA, etc
Thyroid, DHEA, Pregnenolone, Progesterone, etc
What effect does TRT have on cortisol?
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<blockquote data-quote="Vettester Chris" data-source="post: 14259" data-attributes="member: 696"><p>Agree, very good chance the imbalance does stem to the adrenals. A few steps will be needed to check if it's primary or secondary; will be anxious to see your ACTH results.</p><p></p><p>Yeah, going off the TSH lab by itself to make any determination on the thyroid is a mistake that many physicians make. Easy example ... A patient might have hypothyroidism, whereas Total T4 and Free T4 are suboptimal of lab reference range, say 20%. However, various conditions with the "adrenals", iron, electrolytes, ..., can effect Free T3 from getting into the system. If FT3 isn't adequately getting into the cells, it will build-up, better known as "pooling". This is where Reverse T3 also comes into the picture ... The TSH lab can be influenced by the buildup of FT3, basically tricking the feedback loop and pituitary to look as if thyroid hormone levels are normal.</p><p></p><p>That's just one common scenario seen quite a bit in both men and women. I've seen it with women where their TSH is tanked in the 0.30 range, with evident overt hypothyroidism. This can be problematic with estrogen dominance during menopause, which in the absence of progesterone is nothing less than a train wreck from hell on the adrenals!! </p><p></p><p>When you get a better handle on the adrenals, look into the following labs: TSH, Free T4, Free T3, Reverse T3, TPOab and TgAb on the antibodies. Get both antibodies, as autoimmune disorders can be enzyme or protein specific with the thyroid gland.</p></blockquote><p></p>
[QUOTE="Vettester Chris, post: 14259, member: 696"] Agree, very good chance the imbalance does stem to the adrenals. A few steps will be needed to check if it's primary or secondary; will be anxious to see your ACTH results. Yeah, going off the TSH lab by itself to make any determination on the thyroid is a mistake that many physicians make. Easy example ... A patient might have hypothyroidism, whereas Total T4 and Free T4 are suboptimal of lab reference range, say 20%. However, various conditions with the "adrenals", iron, electrolytes, ..., can effect Free T3 from getting into the system. If FT3 isn't adequately getting into the cells, it will build-up, better known as "pooling". This is where Reverse T3 also comes into the picture ... The TSH lab can be influenced by the buildup of FT3, basically tricking the feedback loop and pituitary to look as if thyroid hormone levels are normal. That's just one common scenario seen quite a bit in both men and women. I've seen it with women where their TSH is tanked in the 0.30 range, with evident overt hypothyroidism. This can be problematic with estrogen dominance during menopause, which in the absence of progesterone is nothing less than a train wreck from hell on the adrenals!! When you get a better handle on the adrenals, look into the following labs: TSH, Free T4, Free T3, Reverse T3, TPOab and TgAb on the antibodies. Get both antibodies, as autoimmune disorders can be enzyme or protein specific with the thyroid gland. [/QUOTE]
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Thyroid, Pregnenolone, Progesterone, DHEA, etc
Thyroid, DHEA, Pregnenolone, Progesterone, etc
What effect does TRT have on cortisol?
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