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Clinical Use of Anabolics and Hormones
Clinical Use of Anabolics and Hormones
TESTOSTERONE AND ANABOLICS MISUSE AND ABUSE
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<blockquote data-quote="madman" data-source="post: 194577" data-attributes="member: 13851"><p><strong>Figure 2 – Pathways of testosterone action through direct interaction with the androgen receptor as well as through its bioactive metabolites, dihydrotestosterone (DHT) and estradiol. Dihydrotestosterone is a more potent, pure androgen that operates through the amplification pathway by interacting with the androgen receptor. Estradiol, the most potent estrogen, operates through the diversification pathway that modifies testosterone tissue effects to act on the estrogen receptor. Both active testosterone metabolites are mostly produced in androgen target tissues that express either 5α reductase or aromatase enzymes as mechanisms for local paracrine modulation of testosterone tissue effects.</strong></p><p>[ATTACH=full]12579[/ATTACH]</p></blockquote><p></p>
[QUOTE="madman, post: 194577, member: 13851"] [B]Figure 2 – Pathways of testosterone action through direct interaction with the androgen receptor as well as through its bioactive metabolites, dihydrotestosterone (DHT) and estradiol. Dihydrotestosterone is a more potent, pure androgen that operates through the amplification pathway by interacting with the androgen receptor. Estradiol, the most potent estrogen, operates through the diversification pathway that modifies testosterone tissue effects to act on the estrogen receptor. Both active testosterone metabolites are mostly produced in androgen target tissues that express either 5α reductase or aromatase enzymes as mechanisms for local paracrine modulation of testosterone tissue effects.[/B] [ATTACH type="full"]12579[/ATTACH] [/QUOTE]
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Clinical Use of Anabolics and Hormones
Clinical Use of Anabolics and Hormones
TESTOSTERONE AND ANABOLICS MISUSE AND ABUSE
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