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Testosterone Replacement, Low T, HCG, & Beyond
When Testosterone Is Not Enough
Targeting of PDE5 for Preservation of Penile Health
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<blockquote data-quote="madman" data-source="post: 218506" data-attributes="member: 13851"><p><strong>Figure 3. <u>Schematic diagram of the nitric oxide (NO) signaling pathway in the penis in the context of recurrent ischemic priapism</u>. The diagram depicts a disturbed regulatory balance that predisposes to excessive penile erection. The pathophysiology features low constitutive NO bioactivity basally and dysregulated phosphodiesterase type 5. Upon periodic heightened NO generation and release, 39,59-cyclic guanosine monophosphate is not degraded and accumulates, causing an excessive erectile tissue vasorelaxant response.</strong></p><p><strong>[ATTACH=full]19958[/ATTACH]</strong></p></blockquote><p></p>
[QUOTE="madman, post: 218506, member: 13851"] [B]Figure 3. [U]Schematic diagram of the nitric oxide (NO) signaling pathway in the penis in the context of recurrent ischemic priapism[/U]. The diagram depicts a disturbed regulatory balance that predisposes to excessive penile erection. The pathophysiology features low constitutive NO bioactivity basally and dysregulated phosphodiesterase type 5. Upon periodic heightened NO generation and release, 39,59-cyclic guanosine monophosphate is not degraded and accumulates, causing an excessive erectile tissue vasorelaxant response. [ATTACH type="full"]19958[/ATTACH][/B] [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
When Testosterone Is Not Enough
Targeting of PDE5 for Preservation of Penile Health
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