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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone and Men's Health Articles
Significant decrease in SHBG with the use of Oral TU (JATENZO)
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<blockquote data-quote="Cataceous" data-source="post: 221587" data-attributes="member: 38109"><p>According to Wiki, NAFLD includes NASH: "The term NAFLD encompasses a continuum of liver abnormalities, from non-alcoholic fatty liver (NAFL, simple steatosis) to non-alcoholic steatohepatitis (NASH)." Also confusing is that occasionally NAFLD progresses to NASH. Does the inverse correlation with SHBG not exist in this subset from the beginning, or does it exist and flip during the progression? "One debated mechanism proposes that hepatic steatosis progresses to steatosis with inflammation following some further injury, or <em>second hit</em>. Oxidative stress, hormonal imbalances, and mitochondrial abnormalities are potential causes of this "second hit" phenomenon."[<a href="https://en.wikipedia.org/wiki/Non-alcoholic_fatty_liver_disease#Pathophysiology" target="_blank">R</a>]</p></blockquote><p></p>
[QUOTE="Cataceous, post: 221587, member: 38109"] According to Wiki, NAFLD includes NASH: "The term NAFLD encompasses a continuum of liver abnormalities, from non-alcoholic fatty liver (NAFL, simple steatosis) to non-alcoholic steatohepatitis (NASH)." Also confusing is that occasionally NAFLD progresses to NASH. Does the inverse correlation with SHBG not exist in this subset from the beginning, or does it exist and flip during the progression? "One debated mechanism proposes that hepatic steatosis progresses to steatosis with inflammation following some further injury, or [I]second hit[/I]. Oxidative stress, hormonal imbalances, and mitochondrial abnormalities are potential causes of this "second hit" phenomenon."[[URL='https://en.wikipedia.org/wiki/Non-alcoholic_fatty_liver_disease#Pathophysiology']R[/URL]] [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone and Men's Health Articles
Significant decrease in SHBG with the use of Oral TU (JATENZO)
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