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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Basics & Questions
Short Course: Pathology of the Endocrine Module (Axis)
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<blockquote data-quote="Re-Ride" data-source="post: 35724" data-attributes="member: 8395"><p><a href="https://www.uthsc.edu/endocrinology/documents/ch08-syllabus-Childress.pdf" target="_blank">https://www.uthsc.edu/endocrinology/documents/ch08-syllabus-Childress.pdf</a></p><p></p><p>This 2010 course intended for UT health sciences students of Dale Childress MD is a decent reference material and a fairly easy read for the advanced layman.</p><p></p><p>excerpt:</p><p></p><p>'Both neural input from the central nervous system and humoral factors from the testis modulate the secretion of GnRH.</p><p></p><p>The GnRH neurons receive</p><p></p><p>input from neurons in other parts of the brain including the amygdala and both the</p><p>olfactory and the visual cortex. The release of GnRH is seasonal (peaks in the spring),</p><p></p><p>circadian (highest testosterone levels are in the a.m.) and pulsatile (peaks occur every</p><p>90-120 minutes). GnRH has a very short half</p><p>-life in the blood (approximately 2 to 5</p><p>minutes). The pituitary gland is therefore exposed to hi</p><p>gh levels of GnRH in</p><p>hypophyseal</p><p>-portal blood for brief periods of time. This pulsatile pattern of GnRH</p><p>release appears to be essential for stimulatory effects on LH and FSH release whereas</p><p></p><p>constant exposure to GnRH results in paradoxical inhibitory effect</p><p>s on LH and FSH</p><p>release. "</p></blockquote><p></p>
[QUOTE="Re-Ride, post: 35724, member: 8395"] [URL]https://www.uthsc.edu/endocrinology/documents/ch08-syllabus-Childress.pdf[/URL] This 2010 course intended for UT health sciences students of Dale Childress MD is a decent reference material and a fairly easy read for the advanced layman. excerpt: 'Both neural input from the central nervous system and humoral factors from the testis modulate the secretion of GnRH. The GnRH neurons receive input from neurons in other parts of the brain including the amygdala and both the olfactory and the visual cortex. The release of GnRH is seasonal (peaks in the spring), circadian (highest testosterone levels are in the a.m.) and pulsatile (peaks occur every 90-120 minutes). GnRH has a very short half -life in the blood (approximately 2 to 5 minutes). The pituitary gland is therefore exposed to hi gh levels of GnRH in hypophyseal -portal blood for brief periods of time. This pulsatile pattern of GnRH release appears to be essential for stimulatory effects on LH and FSH release whereas constant exposure to GnRH results in paradoxical inhibitory effect s on LH and FSH release. " [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Basics & Questions
Short Course: Pathology of the Endocrine Module (Axis)
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