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Testosterone Replacement, Low T, HCG, & Beyond
Clomid for PCT, fertility or low T
Pituitary restart while on TRT: promising initial results with GnRH plus enclomiphene
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<blockquote data-quote="Cataceous" data-source="post: 222476" data-attributes="member: 38109"><p>I've been intending to review these measurements, so your question can provide the impetus: LH and FSH have bounced around, primarily in response to sub-experiments. Peak values were obtained about 5-6 months in, with LH at 2.2 mIU/mL (1.7-8.6) and FSH at 1.7 mIU/mL (1.5-12.4). These reference ranges apply throughout. The next test yielded 2.1 and 1.4 respectively, so there are three similars sets of measurements representing steady state with 12.5 mg enclomiphene daily along with 5 mg of testosterone in a TP/TE blend.</p><p></p><p>After that enclomiphene was discontinued for a month, while testosterone intake was reduced to 3.8 mg/day. LH and FSH dropped to 1.5 mIU/mL and 0.8 mIU/mL respectively. As documented previously, this sub-experiment was ended due to unrelenting testicular discomfort.</p><p></p><p>Next, enclomiphene was resumed at the lower rate of 12.5 mg EOD. Testosterone was increased to 4.4 mg/day. After six months LH and FSH were 1.5 mIU/mL and 0.9 mIU/mL respectively, suggesting a dose-dependent response to enclomiphene.</p><p></p><p>The next experiment used 3.8 mg of testosterone daily from testosterone propionate, along with 12.5 enclomiphene E4D. After three months of this LH was below the detection threshold. It seems that the lower troughs of pure propionate were not enough to compensate for the reduced enclomiphene.</p><p></p><p>EOD dosing of enclomiphene was resumed along with 4.4 mg testosterone daily from the TP/TE blend. After three months of this enclomiphene was increased to 12.5 mg daily. Two months later, bringing us almost to the present day, LH had recovered to 1.3 mIU/mL, still below the expected steady state level of about 2 mIU/mL.</p></blockquote><p></p>
[QUOTE="Cataceous, post: 222476, member: 38109"] I've been intending to review these measurements, so your question can provide the impetus: LH and FSH have bounced around, primarily in response to sub-experiments. Peak values were obtained about 5-6 months in, with LH at 2.2 mIU/mL (1.7-8.6) and FSH at 1.7 mIU/mL (1.5-12.4). These reference ranges apply throughout. The next test yielded 2.1 and 1.4 respectively, so there are three similars sets of measurements representing steady state with 12.5 mg enclomiphene daily along with 5 mg of testosterone in a TP/TE blend. After that enclomiphene was discontinued for a month, while testosterone intake was reduced to 3.8 mg/day. LH and FSH dropped to 1.5 mIU/mL and 0.8 mIU/mL respectively. As documented previously, this sub-experiment was ended due to unrelenting testicular discomfort. Next, enclomiphene was resumed at the lower rate of 12.5 mg EOD. Testosterone was increased to 4.4 mg/day. After six months LH and FSH were 1.5 mIU/mL and 0.9 mIU/mL respectively, suggesting a dose-dependent response to enclomiphene. The next experiment used 3.8 mg of testosterone daily from testosterone propionate, along with 12.5 enclomiphene E4D. After three months of this LH was below the detection threshold. It seems that the lower troughs of pure propionate were not enough to compensate for the reduced enclomiphene. EOD dosing of enclomiphene was resumed along with 4.4 mg testosterone daily from the TP/TE blend. After three months of this enclomiphene was increased to 12.5 mg daily. Two months later, bringing us almost to the present day, LH had recovered to 1.3 mIU/mL, still below the expected steady state level of about 2 mIU/mL. [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Clomid for PCT, fertility or low T
Pituitary restart while on TRT: promising initial results with GnRH plus enclomiphene
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