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Testosterone Replacement, Low T, HCG, & Beyond
Clomid for PCT, fertility or low T
Pituitary restart while on TRT: promising initial results with GnRH plus enclomiphene
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<blockquote data-quote="Cataceous" data-source="post: 207021" data-attributes="member: 38109"><p>I am still happy with the protocol, though I continue to experiment. I've been using the gonadorelin/enclomiphene combination for about 19 months. Kisspeptin-10 has been in the mix for a few months less, and I haven't used it without gonadorelin to see if it is stimulating production of endogenous GnRH. It sounds like that's what you're interested in. It would be good to know if this works.</p><p></p><p>From what I've read it may work if you can figure out the correct dosing. Enclomiphene is potent enough to keep the pituitary active in spite of normal levels of estradiol. Additionally, exogenous kisspeptin cannot be attenuated by negative feedback from either estrogens or androgens. One caveat is that levels of GnIH cannot be too high. This hormone acts in opposition to kisspeptin and GnRH, making it an HPTA suppressor. One of its functions is to suppress reproductive ability during times of reduced calorie intake. It's easy to imagine that dysfunction in this mechanism is one of various underlying causes of hypogonadism.</p><p></p><p>Your first measure of progress should be in increasing LH/FSH. At least for me it took a couple months to see significant effects.</p></blockquote><p></p>
[QUOTE="Cataceous, post: 207021, member: 38109"] I am still happy with the protocol, though I continue to experiment. I've been using the gonadorelin/enclomiphene combination for about 19 months. Kisspeptin-10 has been in the mix for a few months less, and I haven't used it without gonadorelin to see if it is stimulating production of endogenous GnRH. It sounds like that's what you're interested in. It would be good to know if this works. From what I've read it may work if you can figure out the correct dosing. Enclomiphene is potent enough to keep the pituitary active in spite of normal levels of estradiol. Additionally, exogenous kisspeptin cannot be attenuated by negative feedback from either estrogens or androgens. One caveat is that levels of GnIH cannot be too high. This hormone acts in opposition to kisspeptin and GnRH, making it an HPTA suppressor. One of its functions is to suppress reproductive ability during times of reduced calorie intake. It's easy to imagine that dysfunction in this mechanism is one of various underlying causes of hypogonadism. Your first measure of progress should be in increasing LH/FSH. At least for me it took a couple months to see significant effects. [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Clomid for PCT, fertility or low T
Pituitary restart while on TRT: promising initial results with GnRH plus enclomiphene
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