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Clinical Use of Anabolics and Hormones
Clinical Use of Anabolics and Hormones
Oxandrolone with 10mg DHEA at the end: blood tests and my experience
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<blockquote data-quote="Rock H. Johnson" data-source="post: 174142" data-attributes="member: 39049"><p>The only mild anabolic that is not reducing endogenous testosterone is Beta Ecdysterone because surprisingly it is acting on Estrogen Receptors and allegedly slightly reduces estrogen levels in males. </p><p></p><p>All of the research confirms these effects in animals, in humans not that much, tho.</p><p>There is not much evidence beyond <em>in vitro</em> to suggest ecdysterone useful for muscle protein synthesis or strength gains.</p><p></p><p>[URL unfurl="true"]https://www.ncbi.nlm.nih.gov/pubmed/18500969[/URL]</p><p><span style="font-size: 26px"><strong>Effects of methoxyisoflavone, ecdysterone, and sulfo-polysaccharide supplementation on training adaptations in resistance-trained males.</strong></span></p><p><a href="https://www.ncbi.nlm.nih.gov/pubmed/?term=Wilborn%20CD%5BAuthor%5D&cauthor=true&cauthor_uid=18500969" target="_blank">Wilborn CD</a>1, <a href="https://www.ncbi.nlm.nih.gov/pubmed/?term=Taylor%20LW%5BAuthor%5D&cauthor=true&cauthor_uid=18500969" target="_blank">Taylor LW</a>, <a href="https://www.ncbi.nlm.nih.gov/pubmed/?term=Campbell%20BI%5BAuthor%5D&cauthor=true&cauthor_uid=18500969" target="_blank">Campbell BI</a>, <a href="https://www.ncbi.nlm.nih.gov/pubmed/?term=Kerksick%20C%5BAuthor%5D&cauthor=true&cauthor_uid=18500969" target="_blank">Kerksick C</a>, <a href="https://www.ncbi.nlm.nih.gov/pubmed/?term=Rasmussen%20CJ%5BAuthor%5D&cauthor=true&cauthor_uid=18500969" target="_blank">Rasmussen CJ</a>, <a href="https://www.ncbi.nlm.nih.gov/pubmed/?term=Greenwood%20M%5BAuthor%5D&cauthor=true&cauthor_uid=18500969" target="_blank">Greenwood M</a>, <a href="https://www.ncbi.nlm.nih.gov/pubmed/?term=Kreider%20RB%5BAuthor%5D&cauthor=true&cauthor_uid=18500969" target="_blank">Kreider RB</a>.</p><p><span style="font-size: 18px"><strong><a href="https://www.ncbi.nlm.nih.gov/pubmed/18500969#" target="_blank">Author information</a></strong></span></p><p><span style="font-size: 18px"><strong></strong></span></p><p><span style="font-size: 18px"><strong>Abstract</strong></span></p><p><strong>PURPOSE:</strong></p><p>Methoxyisoflavone (M), 20-hydroxyecdysone (E), and sulfo-polysaccharide (CSP3) have been marketed to athletes as dietary supplements that can increase strength and muscle mass during resistance-training. However, little is known about their potential ergogenic value. The purpose of this study was to determine whether these supplements affect training adaptations and/or markers of muscle anabolism/catabolism in resistance-trained athletes.</p><p><strong>METHODS:</strong></p><p>Forty-five resistance-trained males (20.5 +/- 3 yrs; 179 +/- 7 cm, 84 +/- 16 kg, 17.3 +/- 9% body fat) were matched according to FFM and randomly assigned to ingest in a double blind manner supplements containing either a placebo (P); 800 mg/day of M; 200 mg of E; or, 1,000 mg/day of CSP3 for 8-weeks during training. At 0, 4, and 8-weeks, subjects donated fasting blood samples and completed comprehensive muscular strength, muscular endurance, anaerobic capacity, and body composition analysis. Data were analyzed by repeated measures ANOVA.</p><p><strong>RESULTS:</strong></p><p>No significant differences (p > 0.05) were observed in training adaptations among groups in the variables FFM, percent body fat, bench press 1 RM, leg press 1 RM or sprint peak power. Anabolic/catabolic analysis revealed no significant differences among groups in active testosterone (AT), free testosterone (FT), cortisol, the AT to cortisol ratio, urea nitrogen, creatinine, the blood urea nitrogen to creatinine ratio. In addition, no significant differences were seen from pre to post supplementation and/or training in AT, FT, or cortisol.</p><p><strong>CONCLUSION:</strong></p><p>Results indicate that M, E, and CSP3 supplementation do not affect body composition or training adaptations nor do they influence the anabolic/catabolic hormone status or general markers of catabolism in resistance-trained males.</p></blockquote><p></p>
[QUOTE="Rock H. Johnson, post: 174142, member: 39049"] The only mild anabolic that is not reducing endogenous testosterone is Beta Ecdysterone because surprisingly it is acting on Estrogen Receptors and allegedly slightly reduces estrogen levels in males. All of the research confirms these effects in animals, in humans not that much, tho. There is not much evidence beyond [I]in vitro[/I] to suggest ecdysterone useful for muscle protein synthesis or strength gains. [URL unfurl="true"]https://www.ncbi.nlm.nih.gov/pubmed/18500969[/URL] [SIZE=26px][B]Effects of methoxyisoflavone, ecdysterone, and sulfo-polysaccharide supplementation on training adaptations in resistance-trained males.[/B][/SIZE] [URL='https://www.ncbi.nlm.nih.gov/pubmed/?term=Wilborn%20CD%5BAuthor%5D&cauthor=true&cauthor_uid=18500969']Wilborn CD[/URL]1, [URL='https://www.ncbi.nlm.nih.gov/pubmed/?term=Taylor%20LW%5BAuthor%5D&cauthor=true&cauthor_uid=18500969']Taylor LW[/URL], [URL='https://www.ncbi.nlm.nih.gov/pubmed/?term=Campbell%20BI%5BAuthor%5D&cauthor=true&cauthor_uid=18500969']Campbell BI[/URL], [URL='https://www.ncbi.nlm.nih.gov/pubmed/?term=Kerksick%20C%5BAuthor%5D&cauthor=true&cauthor_uid=18500969']Kerksick C[/URL], [URL='https://www.ncbi.nlm.nih.gov/pubmed/?term=Rasmussen%20CJ%5BAuthor%5D&cauthor=true&cauthor_uid=18500969']Rasmussen CJ[/URL], [URL='https://www.ncbi.nlm.nih.gov/pubmed/?term=Greenwood%20M%5BAuthor%5D&cauthor=true&cauthor_uid=18500969']Greenwood M[/URL], [URL='https://www.ncbi.nlm.nih.gov/pubmed/?term=Kreider%20RB%5BAuthor%5D&cauthor=true&cauthor_uid=18500969']Kreider RB[/URL]. [SIZE=18px][B][URL='https://www.ncbi.nlm.nih.gov/pubmed/18500969#']Author information[/URL] Abstract[/B][/SIZE] [B]PURPOSE:[/B] Methoxyisoflavone (M), 20-hydroxyecdysone (E), and sulfo-polysaccharide (CSP3) have been marketed to athletes as dietary supplements that can increase strength and muscle mass during resistance-training. However, little is known about their potential ergogenic value. The purpose of this study was to determine whether these supplements affect training adaptations and/or markers of muscle anabolism/catabolism in resistance-trained athletes. [B]METHODS:[/B] Forty-five resistance-trained males (20.5 +/- 3 yrs; 179 +/- 7 cm, 84 +/- 16 kg, 17.3 +/- 9% body fat) were matched according to FFM and randomly assigned to ingest in a double blind manner supplements containing either a placebo (P); 800 mg/day of M; 200 mg of E; or, 1,000 mg/day of CSP3 for 8-weeks during training. At 0, 4, and 8-weeks, subjects donated fasting blood samples and completed comprehensive muscular strength, muscular endurance, anaerobic capacity, and body composition analysis. Data were analyzed by repeated measures ANOVA. [B]RESULTS:[/B] No significant differences (p > 0.05) were observed in training adaptations among groups in the variables FFM, percent body fat, bench press 1 RM, leg press 1 RM or sprint peak power. Anabolic/catabolic analysis revealed no significant differences among groups in active testosterone (AT), free testosterone (FT), cortisol, the AT to cortisol ratio, urea nitrogen, creatinine, the blood urea nitrogen to creatinine ratio. In addition, no significant differences were seen from pre to post supplementation and/or training in AT, FT, or cortisol. [B]CONCLUSION:[/B] Results indicate that M, E, and CSP3 supplementation do not affect body composition or training adaptations nor do they influence the anabolic/catabolic hormone status or general markers of catabolism in resistance-trained males. [/QUOTE]
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Clinical Use of Anabolics and Hormones
Clinical Use of Anabolics and Hormones
Oxandrolone with 10mg DHEA at the end: blood tests and my experience
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