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Thyroid, Pregnenolone, Progesterone, DHEA, etc
Thyroid, DHEA, Pregnenolone, Progesterone, etc
New Thyroid numbers
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<blockquote data-quote="Vettester Chris" data-source="post: 89508" data-attributes="member: 696"><p>Pooling is the result of T3 not getting transported effectively into the body and utilized into the cells. Several variables need to be in play, primarily iron, ferritin, cortisol, and in some respects D3, magnesium and other electrolytes, in order for T3 to be fully effective. The body will 'usually' shift it's conversion and production of Reverse T3, when imbalances are evident (causing FT3 to pool), and/or other issues (sickness, injury, pathology, etc.) are evident, which puts the demand on the body to conserve energy. </p><p></p><p>Although, the RT3 serum & RT3/FT3 ratios are good markers to help assess pooling, it doesn't mean that pooling can't happen if the RT3 picture looks normal. Keep in mind, T4 converts down to T3, and it also converts to RT3. When problems occur, the body will shift the T4 conversion (T4 to T3) to higher rates of RT3 (T4 to RT3). It makes sense, as it's one of the body's way to converse energy and keep low until things are better ... BUT, if T4 levels are low to begin with, then obviously that will be relative to the amount actual RT3 being converted downstream.</p><p></p><p>To treat pooling, you need to identify the 'cause' of what's preventing T3 from reaching the cells. In Vince Carter's case, the RT3/FT3 ratio is 22, which fine, but FT4 is at the low end of the reference range, and FT3 is more towards the top. I lean with Ratbag on the fact that hypothyroidism is almost a given when ferritin is that low.</p></blockquote><p></p>
[QUOTE="Vettester Chris, post: 89508, member: 696"] Pooling is the result of T3 not getting transported effectively into the body and utilized into the cells. Several variables need to be in play, primarily iron, ferritin, cortisol, and in some respects D3, magnesium and other electrolytes, in order for T3 to be fully effective. The body will 'usually' shift it's conversion and production of Reverse T3, when imbalances are evident (causing FT3 to pool), and/or other issues (sickness, injury, pathology, etc.) are evident, which puts the demand on the body to conserve energy. Although, the RT3 serum & RT3/FT3 ratios are good markers to help assess pooling, it doesn't mean that pooling can't happen if the RT3 picture looks normal. Keep in mind, T4 converts down to T3, and it also converts to RT3. When problems occur, the body will shift the T4 conversion (T4 to T3) to higher rates of RT3 (T4 to RT3). It makes sense, as it's one of the body's way to converse energy and keep low until things are better ... BUT, if T4 levels are low to begin with, then obviously that will be relative to the amount actual RT3 being converted downstream. To treat pooling, you need to identify the 'cause' of what's preventing T3 from reaching the cells. In Vince Carter's case, the RT3/FT3 ratio is 22, which fine, but FT4 is at the low end of the reference range, and FT3 is more towards the top. I lean with Ratbag on the fact that hypothyroidism is almost a given when ferritin is that low. [/QUOTE]
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Thyroid, Pregnenolone, Progesterone, DHEA, etc
Thyroid, DHEA, Pregnenolone, Progesterone, etc
New Thyroid numbers
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