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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
New Therapies for Male Androgenetic Alopecia?
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<blockquote data-quote="madman" data-source="post: 234942" data-attributes="member: 13851"><p><em><strong>*AGA is characterized by a decrease in hair density in androgenetic areas of the scalp due to progressive miniaturization of the hair follicle. The etiopathogenesis is multifactorial and complex</strong></em></p><p><em><strong></strong></em></p><p> <em><strong><em><strong>*The activity of the 5-alpha-reductase (5-AR) enzyme converts free testosterone into 5-alpha-dihydrotestosterone. Later, it binds to the androgen receptor in the dermal papilla of the hair follicle and activates the genes responsible for gradual hair loss; this occurs in genetically susceptible people. After several hair cycles, the duration of the anagen phase shortens, and the matrix size decreases, resulting in clinically evident miniaturized hairs </strong></em></strong></em></p><p><em><strong></strong></em></p><p><em><strong>*Oral finasteride, which inhibits the type II enzyme, is a US FDA-approved drug to treat AGA, while oral dutasteride inhibits both types I and II enzymes and has already been approved for the treatment of AGA in Japan and South Korea</strong></em></p><p></p><p><strong>*</strong><em><strong>According to the published evidence, oral dutasteride and oral minoxidil are probably some of the most effective medical treatments available. However, their use is limited as neither are FDA-approved options for hair loss</strong></em></p></blockquote><p></p>
[QUOTE="madman, post: 234942, member: 13851"] [I][B]*AGA is characterized by a decrease in hair density in androgenetic areas of the scalp due to progressive miniaturization of the hair follicle. The etiopathogenesis is multifactorial and complex [I][B]*The activity of the 5-alpha-reductase (5-AR) enzyme converts free testosterone into 5-alpha-dihydrotestosterone. Later, it binds to the androgen receptor in the dermal papilla of the hair follicle and activates the genes responsible for gradual hair loss; this occurs in genetically susceptible people. After several hair cycles, the duration of the anagen phase shortens, and the matrix size decreases, resulting in clinically evident miniaturized hairs [/B][/I] *Oral finasteride, which inhibits the type II enzyme, is a US FDA-approved drug to treat AGA, while oral dutasteride inhibits both types I and II enzymes and has already been approved for the treatment of AGA in Japan and South Korea[/B][/I] [B]*[/B][I][B]According to the published evidence, oral dutasteride and oral minoxidil are probably some of the most effective medical treatments available. However, their use is limited as neither are FDA-approved options for hair loss[/B][/I] [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
New Therapies for Male Androgenetic Alopecia?
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