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New labs. Not sure where to go from here? Need advice.
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<blockquote data-quote="Cataceous" data-source="post: 196918" data-attributes="member: 38109"><p>It does surprise me that your pretty hefty dose of T3 isn't knocking down rT3 better. It probably is a good idea to try a smaller dose of the normal-release form. As I recall, for me 10 mcg liothyronine cut rT3 in half, from around 23 ng/dL. This did seem to resolve my problem with later-day fatigue.</p><p></p><p>I've also had low progesterone and prolactin creeping up, and I've had all the symptoms you list. What worked for me to resolve these issues is easy to state, but potentially complex to implement: I try to make the hormones under my control look like those of the <em>average</em> healthy young man. It's not a perfect substitute for knowing exactly what's optimal, but it's reasonable if you consider evolutionary forces selecting levels that are more likely to make an individual be successful in life. Thus being average can be a good thing: although the term "average looks" is considered somewhat derogatory, the reality is that averaging the facial features of numerous individuals results in a composite that ranks high on appearance. It makes sense when you stop to think about it: faces that are excessively narrow or wide detract from appearance. So the corresponding argument for hormones is that if something is extreme, like testosterone > 1,500 ng/dL, then there's probably a price to be paid.</p><p></p><p>The easiest adjustments are supplementing with progesterone and reducing testosterone—normalizing levels in each case. Sky-high DHT may mean a return to injections. A little more complicated is restoring diurnal variation to testosterone. Do I know this is helpful? Not for sure. But does natural do it for no reason? If all goes well these changes also result in normalization of the downstream hormones, DHT and estradiol. At this point it may be necessary to look upstream to resolve remaining problems. HCG is used to replace the missing LH, with varying degrees of success. The vastly different half-lives is a significant issue. More speculative is that TRT's suppression of GnRH and kisspeptin can create problems with libido and sexual function. Unfortunately research is lacking in this area.</p></blockquote><p></p>
[QUOTE="Cataceous, post: 196918, member: 38109"] It does surprise me that your pretty hefty dose of T3 isn't knocking down rT3 better. It probably is a good idea to try a smaller dose of the normal-release form. As I recall, for me 10 mcg liothyronine cut rT3 in half, from around 23 ng/dL. This did seem to resolve my problem with later-day fatigue. I've also had low progesterone and prolactin creeping up, and I've had all the symptoms you list. What worked for me to resolve these issues is easy to state, but potentially complex to implement: I try to make the hormones under my control look like those of the [I]average[/I] healthy young man. It's not a perfect substitute for knowing exactly what's optimal, but it's reasonable if you consider evolutionary forces selecting levels that are more likely to make an individual be successful in life. Thus being average can be a good thing: although the term "average looks" is considered somewhat derogatory, the reality is that averaging the facial features of numerous individuals results in a composite that ranks high on appearance. It makes sense when you stop to think about it: faces that are excessively narrow or wide detract from appearance. So the corresponding argument for hormones is that if something is extreme, like testosterone > 1,500 ng/dL, then there's probably a price to be paid. The easiest adjustments are supplementing with progesterone and reducing testosterone—normalizing levels in each case. Sky-high DHT may mean a return to injections. A little more complicated is restoring diurnal variation to testosterone. Do I know this is helpful? Not for sure. But does natural do it for no reason? If all goes well these changes also result in normalization of the downstream hormones, DHT and estradiol. At this point it may be necessary to look upstream to resolve remaining problems. HCG is used to replace the missing LH, with varying degrees of success. The vastly different half-lives is a significant issue. More speculative is that TRT's suppression of GnRH and kisspeptin can create problems with libido and sexual function. Unfortunately research is lacking in this area. [/QUOTE]
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