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Clinical Use of Anabolics and Hormones
Clinical Use of Anabolics and Hormones
Nandrolone Experiences
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<blockquote data-quote="DS3" data-source="post: 184489" data-attributes="member: 18514"><p>I follow moreplatesmoredates' videos from time to time. I find the self-experiments he does interesting but never assign to much value to his results given the lack of <em>reliability</em> (repeatability within broader groups of people wherein the same results were maintained) <em>generalizability</em> (ability to generalize findings to large audiences) from case studies. </p><p></p><p><em>β</em>1- and <em>β</em>2-<em>adrenergic receptors</em> in the heart is an interesting hypothesis for the rise in BP seen in males using nandrolone. I do not have much knowledge regarding what those receptors do or what role they could play in nandrolone-induced hypertension. However, Dr. Thomas O'Conner's (anabolic doc) explanation of aldosterone being the primary suspect does make sense to me given the concurrent rise in fluid retention seen in men taking nandrolone. </p><p></p><p>Regardless it nandrolone-induced hypertension is caused by changes in the heart's <em>β</em>1- and <em>β</em>2-<em>adrenergic receptors</em> or aldosterone, would it make a difference in the outcome? Would treating this induced hypertension be enhanced by knowing one way or the other? Or would limiting the dosage and length of administration still be the most effective way to combat long-term side effects such as left-ventricular hypertrophy?</p><p></p><p>[MEDIA=youtube]ob0COFEmzO8[/MEDIA]</p></blockquote><p></p>
[QUOTE="DS3, post: 184489, member: 18514"] I follow moreplatesmoredates' videos from time to time. I find the self-experiments he does interesting but never assign to much value to his results given the lack of [I]reliability[/I] (repeatability within broader groups of people wherein the same results were maintained) [I]generalizability[/I] (ability to generalize findings to large audiences) from case studies. [I]β[/I]1- and [I]β[/I]2-[I]adrenergic receptors[/I] in the heart is an interesting hypothesis for the rise in BP seen in males using nandrolone. I do not have much knowledge regarding what those receptors do or what role they could play in nandrolone-induced hypertension. However, Dr. Thomas O'Conner's (anabolic doc) explanation of aldosterone being the primary suspect does make sense to me given the concurrent rise in fluid retention seen in men taking nandrolone. Regardless it nandrolone-induced hypertension is caused by changes in the heart's [I]β[/I]1- and [I]β[/I]2-[I]adrenergic receptors[/I] or aldosterone, would it make a difference in the outcome? Would treating this induced hypertension be enhanced by knowing one way or the other? Or would limiting the dosage and length of administration still be the most effective way to combat long-term side effects such as left-ventricular hypertrophy? [MEDIA=youtube]ob0COFEmzO8[/MEDIA] [/QUOTE]
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Clinical Use of Anabolics and Hormones
Clinical Use of Anabolics and Hormones
Nandrolone Experiences
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