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Clinical Use of Anabolics and Hormones
Clinical Use of Anabolics and Hormones
Nandrolone Experiences
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<blockquote data-quote="DS3" data-source="post: 176497" data-attributes="member: 18514"><p>Dopamine impact libido--> libido precedes the release of nitric oxide and acetylcholine for genital stimulation. Lower dopamine would impact erectile function.</p><p></p><p>[URL unfurl="true"]https://www.ncbi.nlm.nih.gov/pubmed/11805404[/URL]</p><p></p><p>The potential for ED and libido dysfunction while co-administering nandrolone and T is a complicated topic. Due to the fact that co-administration of nandrolone and T is the generally prescribed use of nandrolone and the fact that some men experience ED during that protocol or during nandrolone base use without an aromatizing drug, it can't be inferred that negligible estradiol is to blame for the potential negative effects. You have men who maintain proper E2 during nandrolone & T administration that experience ED and/or lowered libido. Conversely, you have some men who run T & N who experience no ED and report significantly elevated sense of well-being and increased libido. Then, you have men (small group of men that do this) who run a nandrolone base and experience ED with or without adding an aromatizing drug to the protocol. Conversely, you have similar protocols run by other men who claim that this is a 'nectar of the gods' protocol.</p><p></p><p>It can't be as simple as estrogen or the lackthereof, nor does it have to be as complicated as stating that there can be zero common factors. The likelihood that the occurrence of ED and/or lowered libido is a combination of low androgenicity of nandrolone, potential for decreased dopamine, potential for reduced bioavailable nitric oxide, potential for low E2 (if used as a base), and potential other explanations regarding the relationship between progesterone and estrogen are all viable. The likelihood that the impact of these factors, aggregated or separate, express themselves with varying degrees in individual patients would also be a factor and further confound what the common etiology is behind lowered libido and/or ED while using nandrolone in therapeutic settings.</p><p></p><p>I'll tell you what, though. You and I will have more confident answers for these occurrences in the future! Studies will be conducted, hopefully, as nandrolone is being used more frequently as a therapeutic agent.</p></blockquote><p></p>
[QUOTE="DS3, post: 176497, member: 18514"] Dopamine impact libido--> libido precedes the release of nitric oxide and acetylcholine for genital stimulation. Lower dopamine would impact erectile function. [URL unfurl="true"]https://www.ncbi.nlm.nih.gov/pubmed/11805404[/URL] The potential for ED and libido dysfunction while co-administering nandrolone and T is a complicated topic. Due to the fact that co-administration of nandrolone and T is the generally prescribed use of nandrolone and the fact that some men experience ED during that protocol or during nandrolone base use without an aromatizing drug, it can't be inferred that negligible estradiol is to blame for the potential negative effects. You have men who maintain proper E2 during nandrolone & T administration that experience ED and/or lowered libido. Conversely, you have some men who run T & N who experience no ED and report significantly elevated sense of well-being and increased libido. Then, you have men (small group of men that do this) who run a nandrolone base and experience ED with or without adding an aromatizing drug to the protocol. Conversely, you have similar protocols run by other men who claim that this is a 'nectar of the gods' protocol. It can't be as simple as estrogen or the lackthereof, nor does it have to be as complicated as stating that there can be zero common factors. The likelihood that the occurrence of ED and/or lowered libido is a combination of low androgenicity of nandrolone, potential for decreased dopamine, potential for reduced bioavailable nitric oxide, potential for low E2 (if used as a base), and potential other explanations regarding the relationship between progesterone and estrogen are all viable. The likelihood that the impact of these factors, aggregated or separate, express themselves with varying degrees in individual patients would also be a factor and further confound what the common etiology is behind lowered libido and/or ED while using nandrolone in therapeutic settings. I'll tell you what, though. You and I will have more confident answers for these occurrences in the future! Studies will be conducted, hopefully, as nandrolone is being used more frequently as a therapeutic agent. [/QUOTE]
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Clinical Use of Anabolics and Hormones
Clinical Use of Anabolics and Hormones
Nandrolone Experiences
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