ExcelMale
Menu
Home
What's new
Latest activity
Forums
New posts
Search forums
What's new
New posts
Latest activity
Videos
Lab Tests
Doctor Finder
Buy Books
About Us
Men’s Health Coaching
Log in
Register
What's new
Search
Search
Search titles only
By:
New posts
Search forums
Menu
Log in
Register
Navigation
Install the app
Install
More options
Contact us
Close Menu
Forums
Clinical Use of Anabolics and Hormones
Clinical Use of Anabolics and Hormones
Nandrolone Experiences
JavaScript is disabled. For a better experience, please enable JavaScript in your browser before proceeding.
You are using an out of date browser. It may not display this or other websites correctly.
You should upgrade or use an
alternative browser
.
Reply to thread
Message
<blockquote data-quote="DS3" data-source="post: 170779" data-attributes="member: 18514"><p>"What numbers did you get for two hours post-injection? This is closest to when the peak occurs, at least with propionate."</p><p></p><p>"It's well established in the scientific literature that the area under the response curve is proportional to the dose. You don't have nearly enough data points to firmly establish a contradiction. Unfortunately it's not really practical for an individual to get enough data to figure out what's really happening in a case like this."</p><p></p><p>Think of it like this. If at 50 mgs of Prop per day I spike at <strong>1400 ng/dL</strong> (speculative) and after 10 hours drop to <strong>1178 ng/dL</strong> (previously shown), likely dropping somewhere at or below <strong>800 ng/dL</strong> at 24 hours, then it can be logically inferred that 14/24 hours of the day (58.3% of the time) I am within range of TT levels that TRT patients aim to achieve. And if I were to drop my dosage, my spike and trough would be lower, which would not be desirable.</p><p></p><p>"I've seen no credible scientific evidence for big differences in how different esters are metabolized. They all end up in the blood where the esters are quickly cleaved from the testosterone. The only differences are in the speed of release from injected depots. This is again well established, and the extraordinary claim that it's wrong requires more support than a few isolated measurements.</p></blockquote><p>"</p><p></p><p>[USER=38109]@Cataceous[/USER] You seem to be laboring under the delusion that I am seeking validation in my claim that my body metabolizes long-acting esters differently than short-acting esters. After a decade of taking testosterone, I have tried just about every protocol you could think of. With long-acting esters, I experience higher TT and E2, gastrointestinal pain (only experienced with long-acting esters, regardless of dietary intake). With short-acting esters, I can run higher doses without experiencing E2 issues and a quantifiable difference in TT levels (lower) than with long-acting esters (of lower doses). These claims are all quantifiable from my end. </p><p></p><p>Don't labor my friend.</p><p>[/QUOTE]</p>
[QUOTE="DS3, post: 170779, member: 18514"] "What numbers did you get for two hours post-injection? This is closest to when the peak occurs, at least with propionate." "It's well established in the scientific literature that the area under the response curve is proportional to the dose. You don't have nearly enough data points to firmly establish a contradiction. Unfortunately it's not really practical for an individual to get enough data to figure out what's really happening in a case like this." Think of it like this. If at 50 mgs of Prop per day I spike at [B]1400 ng/dL[/B] (speculative) and after 10 hours drop to [B]1178 ng/dL[/B] (previously shown), likely dropping somewhere at or below [B]800 ng/dL[/B] at 24 hours, then it can be logically inferred that 14/24 hours of the day (58.3% of the time) I am within range of TT levels that TRT patients aim to achieve. And if I were to drop my dosage, my spike and trough would be lower, which would not be desirable. "I've seen no credible scientific evidence for big differences in how different esters are metabolized. They all end up in the blood where the esters are quickly cleaved from the testosterone. The only differences are in the speed of release from injected depots. This is again well established, and the extraordinary claim that it's wrong requires more support than a few isolated measurements.[/QUOTE]" [USER=38109]@Cataceous[/USER] You seem to be laboring under the delusion that I am seeking validation in my claim that my body metabolizes long-acting esters differently than short-acting esters. After a decade of taking testosterone, I have tried just about every protocol you could think of. With long-acting esters, I experience higher TT and E2, gastrointestinal pain (only experienced with long-acting esters, regardless of dietary intake). With short-acting esters, I can run higher doses without experiencing E2 issues and a quantifiable difference in TT levels (lower) than with long-acting esters (of lower doses). These claims are all quantifiable from my end. Don't labor my friend. [/QUOTE]
Insert quotes…
Verification
Post reply
Share this page
Facebook
Twitter
Reddit
Pinterest
Tumblr
WhatsApp
Email
Share
Link
Sponsors
Forums
Clinical Use of Anabolics and Hormones
Clinical Use of Anabolics and Hormones
Nandrolone Experiences
This site uses cookies to help personalise content, tailor your experience and to keep you logged in if you register.
By continuing to use this site, you are consenting to our use of cookies.
Accept
Learn more…
Top