ExcelMale
Menu
Home
What's new
Latest activity
Forums
New posts
Search forums
What's new
New posts
Latest activity
Videos
Lab Tests
Doctor Finder
Buy Books
About Us
Men’s Health Coaching
Log in
Register
What's new
Search
Search
Search titles only
By:
New posts
Search forums
Menu
Log in
Register
Navigation
Install the app
Install
More options
Contact us
Close Menu
Forums
Clinical Use of Anabolics and Hormones
Clinical Use of Anabolics and Hormones
My short-term Dbol-only experiment. Or, “How I learned to stop worrying and love oral anabolics”. Part-1
JavaScript is disabled. For a better experience, please enable JavaScript in your browser before proceeding.
You are using an out of date browser. It may not display this or other websites correctly.
You should upgrade or use an
alternative browser
.
Reply to thread
Message
<blockquote data-quote="madman" data-source="post: 274605" data-attributes="member: 13851"><p>First off what does methandrostanonone have to do with testosterone therapy?</p><p></p><p>This is a men's health/HRT forum.</p><p></p><p>You should know better!</p><p></p><p>This is nonsense you should be posting on those other bum*** forums (bum-nation, facepalm, I just don't geddit) littered on the internet.</p><p></p><p>You know the ones loaded with all those blast n cruizers sporting those chemically enhanced fake builds that all those poor young chaps idolize!</p><p></p><p>Real men, they say LMFAO!</p><p></p><p>Other than nandrolone which is used in therapeutic doses for relief/improvement of joint pain or oxandrolone which may be prescribed for the same reason as both are legal compounds methandrostenolone let alone any other AAS is not.</p><p></p><p>Unfortunately, the FDA recently pulled oxandrolone off the market.</p><p></p><p>The only way one can obtain methandrostenolone is through a UGL source.</p><p></p><p>No doctor can legally prescribe such!</p><p></p><p>As you should very well know methandrostenolone could never replace testosterone let alone it is not something that one would use long-term due to the compound being c-17 alpha alkylated which is known to stress the liver.</p><p></p><p>The use of AAS 17α-alkylated orals such as stanozolol, oxandrolone, methyltestosterone, methandrostenolone, oxymetholone, and fluoxymesterone are notorious for driving down HDL, increasing LDL, stressing the liver and hammering down SHBG.</p><p></p><p>The c-17 alpha-alkylated orals put a greater strain on the cardiovascular system and are also known to be liver-toxic when abused.</p><p></p><p>Either way one still needs to be mindful of the time on/off which will be magnified over the years.</p><p></p><p>Even then the dose/duration of use will play a big role.</p><p></p><p>Highly doubtful you are going to run into any serious liver damage from using therapeutic does (oxandrolone/stanazolol) let alone low doses of any other c-17 alpha alkylated orals from short-term use.</p><p></p><p>No AAS would be a testosterone substitution.</p><p></p><p>The main reason testosterone is used for replacement therapy over nandrolone let alone any other AAS is that testosterone drugs <u>provide a hormone that is already produced in the body</u>.</p><p></p><p>More importantly, its metabolites estradiol and DHT are needed in healthy amounts to experience the full spectrum of testosterone-beneficial effects on (cardiovascular health, brain health, libido, erectile function, bone health, tendon health, immune system, lipids, and body composition).</p><p></p><p></p><p><em><strong>*Natural testosterone is viewed as the best androgen for substitution in hypogonadal men. The reason behind the selection is that </strong></em><u><strong><em>testosterone can be converted to DHT and E2, thus developing the full spectrum of testosterone activities in long-term substitution</em></strong></u></p><p></p><p><strong><em>*Preparations of <u>native testosterone or its esters</u> (aromatizable T) should be used for TTh</em></strong></p><p></p><p></p><p></p><p></p><p>Hate to burst your bubble but BJ never relied solely on a 3 week methadrostenolone protocol throughout his career.</p><p></p><p>When he first started dabbling with AAS in the early 80s low dose short-term cycles of T (TP/TS) and methandrostenolone were used.</p><p></p><p>He stopped using methandrostenolone by the mid-80s and the go-to oral which was used in a short-term cyclic fashion for years was stanozolol which was supposedly replaced by Furazabol.</p><p></p><p>Low dose test (TP/TS) was also a staple throughout his and many of the other track and field athletes.</p><p></p><p>Strength/speed/muscular density and recovery was the goal, not excess muscle mass which would have had a negative effect on the athlete's performance.</p><p></p><p>Short-term stints with T/AAS along with a proper diet/training protocol would accomplish this.</p><p></p><p>The goal of such athletes was not to bulk up.</p><p></p><p>Methandrostenolone and oxymetholone would be considered mass-building steroids.</p><p></p><p>Stanozolol and oxandrolone let alone fluoxymesterone would be considered strength steroids and preferred for improving lean mass or athletic performance.</p><p></p><p>These compounds would be staples in strength/speed sports.</p><p></p><p>To take full advantage of T/AAS anabolic potential when it comes to gaining muscle mass one needs to achieve levels well into the supra-physiological range 24/7 in a cyclic fashion typically 6-12 weeks in length.</p><p></p><p>Depending on the T/AAS used 6-12 week cycles are common.</p><p></p><p>The majority of the gains are made in the first 6-8 weeks.</p><p></p><p>To obtain such one would need to use any of the oral AAS dosed daily or spread throughout the day due to the short half-life (PKs) for at least 6 weeks to reap the full benefits.</p><p></p><p>Unesterified injectables such as test suspension would be dosed daily due to the half-life (PK) for 6-8 weeks.</p><p></p><p>Short-acting esterified TP would be dosed daily or EOD due to the half-life (PK) for 6-8 weeks.</p><p></p><p>Esterified medium-acting TC/TE would be dosed once weekly or twice weekly due to the half-life (PK) for 8-12 weeks.</p><p></p><p>Think of how long it takes just to reach steady-state (4-6 weeks) when using the medium-acting esters.</p><p></p><p>No way in hell you are reaping the full anabolic potential of T/AAS using 3-week cycles let alone using low doses even when using the orals.</p><p></p><p>Not if packing on muscle mass is your goal.</p><p></p><p>Put money on it that 5 lbs you put on is not purely dry gains (actin/myosin).</p><p></p><p>Even cycling a low dose of 10 mg daily for 3-week stints is not going to result in throwing on a lot of mass.</p><p></p><p>15-30 mg daily for 6 weeks is where it's at.</p><p></p><p>Even then I would not waste my time with the methandrostenolone if gaining mass was my goal.</p><p></p><p>Would up the ND dose any day to put on some quality muscle before f**king with low dose 3-week stints of methandrostenolone!</p></blockquote><p></p>
[QUOTE="madman, post: 274605, member: 13851"] First off what does methandrostanonone have to do with testosterone therapy? This is a men's health/HRT forum. You should know better! This is nonsense you should be posting on those other bum*** forums (bum-nation, facepalm, I just don't geddit) littered on the internet. You know the ones loaded with all those blast n cruizers sporting those chemically enhanced fake builds that all those poor young chaps idolize! Real men, they say LMFAO! Other than nandrolone which is used in therapeutic doses for relief/improvement of joint pain or oxandrolone which may be prescribed for the same reason as both are legal compounds methandrostenolone let alone any other AAS is not. Unfortunately, the FDA recently pulled oxandrolone off the market. The only way one can obtain methandrostenolone is through a UGL source. No doctor can legally prescribe such! As you should very well know methandrostenolone could never replace testosterone let alone it is not something that one would use long-term due to the compound being c-17 alpha alkylated which is known to stress the liver. The use of AAS 17α-alkylated orals such as stanozolol, oxandrolone, methyltestosterone, methandrostenolone, oxymetholone, and fluoxymesterone are notorious for driving down HDL, increasing LDL, stressing the liver and hammering down SHBG. The c-17 alpha-alkylated orals put a greater strain on the cardiovascular system and are also known to be liver-toxic when abused. Either way one still needs to be mindful of the time on/off which will be magnified over the years. Even then the dose/duration of use will play a big role. Highly doubtful you are going to run into any serious liver damage from using therapeutic does (oxandrolone/stanazolol) let alone low doses of any other c-17 alpha alkylated orals from short-term use. No AAS would be a testosterone substitution. The main reason testosterone is used for replacement therapy over nandrolone let alone any other AAS is that testosterone drugs [U]provide a hormone that is already produced in the body[/U]. More importantly, its metabolites estradiol and DHT are needed in healthy amounts to experience the full spectrum of testosterone-beneficial effects on (cardiovascular health, brain health, libido, erectile function, bone health, tendon health, immune system, lipids, and body composition). [I][B]*Natural testosterone is viewed as the best androgen for substitution in hypogonadal men. The reason behind the selection is that [/B][/I][U][B][I]testosterone can be converted to DHT and E2, thus developing the full spectrum of testosterone activities in long-term substitution[/I][/B][/U] [B][I]*Preparations of [U]native testosterone or its esters[/U] (aromatizable T) should be used for TTh[/I][/B] Hate to burst your bubble but BJ never relied solely on a 3 week methadrostenolone protocol throughout his career. When he first started dabbling with AAS in the early 80s low dose short-term cycles of T (TP/TS) and methandrostenolone were used. He stopped using methandrostenolone by the mid-80s and the go-to oral which was used in a short-term cyclic fashion for years was stanozolol which was supposedly replaced by Furazabol. Low dose test (TP/TS) was also a staple throughout his and many of the other track and field athletes. Strength/speed/muscular density and recovery was the goal, not excess muscle mass which would have had a negative effect on the athlete's performance. Short-term stints with T/AAS along with a proper diet/training protocol would accomplish this. The goal of such athletes was not to bulk up. Methandrostenolone and oxymetholone would be considered mass-building steroids. Stanozolol and oxandrolone let alone fluoxymesterone would be considered strength steroids and preferred for improving lean mass or athletic performance. These compounds would be staples in strength/speed sports. To take full advantage of T/AAS anabolic potential when it comes to gaining muscle mass one needs to achieve levels well into the supra-physiological range 24/7 in a cyclic fashion typically 6-12 weeks in length. Depending on the T/AAS used 6-12 week cycles are common. The majority of the gains are made in the first 6-8 weeks. To obtain such one would need to use any of the oral AAS dosed daily or spread throughout the day due to the short half-life (PKs) for at least 6 weeks to reap the full benefits. Unesterified injectables such as test suspension would be dosed daily due to the half-life (PK) for 6-8 weeks. Short-acting esterified TP would be dosed daily or EOD due to the half-life (PK) for 6-8 weeks. Esterified medium-acting TC/TE would be dosed once weekly or twice weekly due to the half-life (PK) for 8-12 weeks. Think of how long it takes just to reach steady-state (4-6 weeks) when using the medium-acting esters. No way in hell you are reaping the full anabolic potential of T/AAS using 3-week cycles let alone using low doses even when using the orals. Not if packing on muscle mass is your goal. Put money on it that 5 lbs you put on is not purely dry gains (actin/myosin). Even cycling a low dose of 10 mg daily for 3-week stints is not going to result in throwing on a lot of mass. 15-30 mg daily for 6 weeks is where it's at. Even then I would not waste my time with the methandrostenolone if gaining mass was my goal. Would up the ND dose any day to put on some quality muscle before f**king with low dose 3-week stints of methandrostenolone! [/QUOTE]
Insert quotes…
Verification
Post reply
Share this page
Facebook
Twitter
Reddit
Pinterest
Tumblr
WhatsApp
Email
Share
Link
Sponsors
Forums
Clinical Use of Anabolics and Hormones
Clinical Use of Anabolics and Hormones
My short-term Dbol-only experiment. Or, “How I learned to stop worrying and love oral anabolics”. Part-1
This site uses cookies to help personalise content, tailor your experience and to keep you logged in if you register.
By continuing to use this site, you are consenting to our use of cookies.
Accept
Learn more…
Top