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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Basics & Questions
My Ongoing TRT Mystery
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<blockquote data-quote="Simbarn" data-source="post: 240336" data-attributes="member: 44021"><p>Thanks for describing your experience with these blockers. It will help me if I decide to give them another try.</p><p>I have read similar accounts in regard to Yohimbine (as its more of an a2 blocker than a1), so have not tried it. It must have been extremely odd given your normal reaction to anxiety and erectile function!</p><p></p><p>Let me know what happens with your consult with the new doctor and if you do add the Lexapro. 2.5 is certainly a low dose. Why have you chosen this particular SSRI?</p><p>I have used Citalopram (older version of Escitalopram) decades ago. I reacted well to it at the time.</p><p></p><p>My reaction to benzos in regard to sexual function seems to be indicative of a sympathetic dominance issue like you. I also notice generally in the morning when I first wake my erectile/sexual function is much more responsive. After I am up and fully awake, this responsiveness drops markedly.</p><p>My research into nocturnal erections and why they occur is relevant here. It is hypothesised that owing to the almost shut down of the locus coeruleus during REM sleep, the inhibitory mechanisms in the penis are significantly suppressed. Erections therefore occur with great ease during these periods of sleep, smooth muscle in the erectile tissues has a much greater tendency to relax and it is thought that just circulating androgen's are sufficient to stimulate this relaxation during REM sleep, confirming the very direct influence testosterone has on nocturnal erections.</p><p>When one first wakes the body is still partially in this state.</p><p>Do you experience a similar thing in the morning?</p><p></p><p>Testosterone has a very direct effect with nocturnal erections. When it is low these do appear to lessen in frequency and duration.</p><p></p><p>The study you have given me was quite fascinating with regard to SERT binding. It does seem to indicate that androgens could influence this significantly as does estrogen.</p><p>The webinar on hormones and anxiety has certainly given me more to think about, especially given how I think my body has reacted to exogenous testosterone and symapthetic activity, possibly related to this SERT issue. Since adding recombinant hCG back into my protocol I have noticed definite improvements with my issues and this could be related to how important LH is (as mentioned in the webinar) in the conversion of cholesterol into pregnenolone, as many of us already know here.</p><p></p><p>I related to what the doctor said on that webinar describing how he began treating guys for low testosterone and how much time he spent learning about female hormones compared to the minor time spent on male hormones in the late 90's. It was not long after this that I began seeing doctors regarding my testosterone issues! They were clueless!</p></blockquote><p></p>
[QUOTE="Simbarn, post: 240336, member: 44021"] Thanks for describing your experience with these blockers. It will help me if I decide to give them another try. I have read similar accounts in regard to Yohimbine (as its more of an a2 blocker than a1), so have not tried it. It must have been extremely odd given your normal reaction to anxiety and erectile function! Let me know what happens with your consult with the new doctor and if you do add the Lexapro. 2.5 is certainly a low dose. Why have you chosen this particular SSRI? I have used Citalopram (older version of Escitalopram) decades ago. I reacted well to it at the time. My reaction to benzos in regard to sexual function seems to be indicative of a sympathetic dominance issue like you. I also notice generally in the morning when I first wake my erectile/sexual function is much more responsive. After I am up and fully awake, this responsiveness drops markedly. My research into nocturnal erections and why they occur is relevant here. It is hypothesised that owing to the almost shut down of the locus coeruleus during REM sleep, the inhibitory mechanisms in the penis are significantly suppressed. Erections therefore occur with great ease during these periods of sleep, smooth muscle in the erectile tissues has a much greater tendency to relax and it is thought that just circulating androgen's are sufficient to stimulate this relaxation during REM sleep, confirming the very direct influence testosterone has on nocturnal erections. When one first wakes the body is still partially in this state. Do you experience a similar thing in the morning? Testosterone has a very direct effect with nocturnal erections. When it is low these do appear to lessen in frequency and duration. The study you have given me was quite fascinating with regard to SERT binding. It does seem to indicate that androgens could influence this significantly as does estrogen. The webinar on hormones and anxiety has certainly given me more to think about, especially given how I think my body has reacted to exogenous testosterone and symapthetic activity, possibly related to this SERT issue. Since adding recombinant hCG back into my protocol I have noticed definite improvements with my issues and this could be related to how important LH is (as mentioned in the webinar) in the conversion of cholesterol into pregnenolone, as many of us already know here. I related to what the doctor said on that webinar describing how he began treating guys for low testosterone and how much time he spent learning about female hormones compared to the minor time spent on male hormones in the late 90's. It was not long after this that I began seeing doctors regarding my testosterone issues! They were clueless! [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
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My Ongoing TRT Mystery
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