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[USER=38109]@Cataceous[/USER] has got the right idea imo. Higher test and E2 effects many other processes other than HPTA shut down. Even on a low dose of test, LH and FSH are basically gonna be about zero. So I can’t see how a person can be more shut down if they raise their dose. Zero for LH and FSH is zero. It’s not like a person can have their LH and FSH levels less than zero by increasing their doseBut raising androgen levels, and therefore raising E2 and prolactin levels, can definitely effect other processes. I’m pretty sure as u increase ur test dose there’s a possibility of decreasing cortisol levels further, but not 100% positive about that. Just something to consider and look into. I’ve also heard that too high of an E2 level can effect the thyroid negatively. Women have a much greater chance of having thyroid issues than men, and I wonder if it’s due to the fact that they have much higher estrogen levels than males do. Just some other things to consider other than HPTA shut down, when increasing exogenous androgens
[USER=38109]@Cataceous[/USER] has got the right idea imo. Higher test and E2 effects many other processes other than HPTA shut down. Even on a low dose of test, LH and FSH are basically gonna be about zero. So I can’t see how a person can be more shut down if they raise their dose. Zero for LH and FSH is zero. It’s not like a person can have their LH and FSH levels less than zero by increasing their dose
But raising androgen levels, and therefore raising E2 and prolactin levels, can definitely effect other processes. I’m pretty sure as u increase ur test dose there’s a possibility of decreasing cortisol levels further, but not 100% positive about that. Just something to consider and look into. I’ve also heard that too high of an E2 level can effect the thyroid negatively. Women have a much greater chance of having thyroid issues than men, and I wonder if it’s due to the fact that they have much higher estrogen levels than males do. Just some other things to consider other than HPTA shut down, when increasing exogenous androgens
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