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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone and Men's Health Articles
Male sex hormones, aging, and inflammation
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<blockquote data-quote="madman" data-source="post: 242377" data-attributes="member: 13851"><p><strong>Fig. 2 <u>Androgen effects on organ systems</u>. Testosterone and its derivatives have been shown to result in increased muscle protein synthesis (Griggs et al. 1989), amino acid utilization (Shefeld-Moore et al. 1999), and maximum skeletal muscle strength (Bhasin et al. 2001). Androgens have been shown to increase EPO and hematocrit with their effects on CVD still under debate (Shores and Matsumoto 2014). Testosterone increases BMD and osteoblast growth (Nakano et al. 1994) while concurrently decreasing the apoptosis of osteocytes (Kousteni et al. 2001). Testosterone increases T cell maturation (Viselli et al. 1995a, b), decreases the proliferation of autoantibodies (Rettew et al. 2008), as well as signaling involving TLR4, TNF-α, and IL-1β (Corcoran et al. 2010). Androgens decrease the activity of LPL and ACS (Santosa et al. 2017), resulting in a decline in lipogenesis (Weimar et al. 2002). The nervous system responds to testosterone by increasing ant-inflammatory markers including IL-10 (Dalal et al. 1997), remyelination of neurons (Palaszynski et al. 2004), and increasing cognitive and spatial function (Azad et al. 2003; Barrett-Connor et al. 1999; Janowsky et al. 1994). Androgens also decrease the concentration of ROS (Ahlbom et al. 2001) and Aβ plaques in the nervous system (Pike 2001)</strong></p><p><strong>[ATTACH=full]28281[/ATTACH]</strong></p></blockquote><p></p>
[QUOTE="madman, post: 242377, member: 13851"] [B]Fig. 2 [U]Androgen effects on organ systems[/U]. Testosterone and its derivatives have been shown to result in increased muscle protein synthesis (Griggs et al. 1989), amino acid utilization (Shefeld-Moore et al. 1999), and maximum skeletal muscle strength (Bhasin et al. 2001). Androgens have been shown to increase EPO and hematocrit with their effects on CVD still under debate (Shores and Matsumoto 2014). Testosterone increases BMD and osteoblast growth (Nakano et al. 1994) while concurrently decreasing the apoptosis of osteocytes (Kousteni et al. 2001). Testosterone increases T cell maturation (Viselli et al. 1995a, b), decreases the proliferation of autoantibodies (Rettew et al. 2008), as well as signaling involving TLR4, TNF-α, and IL-1β (Corcoran et al. 2010). Androgens decrease the activity of LPL and ACS (Santosa et al. 2017), resulting in a decline in lipogenesis (Weimar et al. 2002). The nervous system responds to testosterone by increasing ant-inflammatory markers including IL-10 (Dalal et al. 1997), remyelination of neurons (Palaszynski et al. 2004), and increasing cognitive and spatial function (Azad et al. 2003; Barrett-Connor et al. 1999; Janowsky et al. 1994). Androgens also decrease the concentration of ROS (Ahlbom et al. 2001) and Aβ plaques in the nervous system (Pike 2001) [ATTACH type="full"]28281[/ATTACH][/B] [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone and Men's Health Articles
Male sex hormones, aging, and inflammation
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