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Testosterone Replacement, Low T, HCG, & Beyond
Clomid for PCT, fertility or low T
Male infertility and gonadotropin treatment
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<blockquote data-quote="madman" data-source="post: 244566" data-attributes="member: 13851"><p><strong>Practice points </strong></p><p></p><p><em><strong>*Empiric hormonal therapy for male infertility patients is widely practiced and relies on the knowledge that FSH- and LH-driven testosterone primarily regulate spermatogenesis</strong></em></p><p><em><strong></strong></em></p><p><em><strong>*Exogenous FSH is the gonadotropin treatment of choice for men with idiopathic oligozoospermia seeking fertility</strong></em></p><p><em><strong></strong></em></p><p><em><strong>*Exogenous FSH therapy might increase sperm quantity in men with idiopathic oligozoospermia, with an apparent positive effect on natural and medically assisted reproduction pregnancy rates</strong></em></p><p><em><strong></strong></em></p><p><em><strong>*Gonadotropin therapy for males with nonobstructive azoospermia might boost intratesticular testosterone production, spermatogenesis, and spermiogenesis, potentially improving sperm retrieval success or the presence of sperm in the ejaculate</strong></em></p><p><em><strong></strong></em></p><p><em><strong>*Evidence from cohort studies and case series suggests that gonadotropin therapy with exogenous hCG and/or FSH could lead to a 10-15% higher sperm retrieval rate than with no treatment in NOA males</strong></em></p><p><em><strong></strong></em></p><p><em><strong>*Based on limited data, the most suitable NOA patients for gonadotropin therapy seem to be hypogonadal men (i.e., serum levels of total testosterone <300 ng/dL) with baseline FSH levels <strong>≤ </strong>12 UI/L and those with histopathology showing maturation arrest (late stages) or hypospermatogenesis</strong></em></p></blockquote><p></p>
[QUOTE="madman, post: 244566, member: 13851"] [B]Practice points [/B] [I][B]*Empiric hormonal therapy for male infertility patients is widely practiced and relies on the knowledge that FSH- and LH-driven testosterone primarily regulate spermatogenesis *Exogenous FSH is the gonadotropin treatment of choice for men with idiopathic oligozoospermia seeking fertility *Exogenous FSH therapy might increase sperm quantity in men with idiopathic oligozoospermia, with an apparent positive effect on natural and medically assisted reproduction pregnancy rates *Gonadotropin therapy for males with nonobstructive azoospermia might boost intratesticular testosterone production, spermatogenesis, and spermiogenesis, potentially improving sperm retrieval success or the presence of sperm in the ejaculate *Evidence from cohort studies and case series suggests that gonadotropin therapy with exogenous hCG and/or FSH could lead to a 10-15% higher sperm retrieval rate than with no treatment in NOA males *Based on limited data, the most suitable NOA patients for gonadotropin therapy seem to be hypogonadal men (i.e., serum levels of total testosterone <300 ng/dL) with baseline FSH levels [B]≤ [/B]12 UI/L and those with histopathology showing maturation arrest (late stages) or hypospermatogenesis[/B][/I] [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Clomid for PCT, fertility or low T
Male infertility and gonadotropin treatment
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