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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
I accidentally took too much Arimidex...did I screw myself!?
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<blockquote data-quote="Vitamin_C" data-source="post: 85157" data-attributes="member: 12373"><p>Do you have labs to indicate you have electrolyte imbalances? DEXA scan to show bone loss or any other lab to show muscle wasting? Do you have labs showing elevated lipids and cholesterol? Elevated prolactin? High CRP? High BP? What do your labs look like pertaining to cortisol and thyroid? Don't just say "they are good".....I'd like to see some actual numbers if you could upload them to the site I'd appreciate it. As a researcher and future practitioner, I am fascinated by these claims but I just see a severe lack of evidence for it. There are women on 1mg of Arimidex a day for sometimes up to 7-10 years, thousands and thousands of women and there is absolutely no data they become insensitive to estrogen via Arimidex. Estrogen is needed for a wide variety of physiological processes in men, from keeping cholesterol in check, prolactin/dopamine etc. I would like to see if you had any lab abnormalities. Like I told nurselyfe, have your endo script you estrogen patches and if you have no high E2 symptoms you can be the first case of this happening in medical history. I am still not understanding the mechanism behind less vascularity and holding water with minuscule amounts of estrogen. Are you having libido/erection problems? Can you show pharmacologically that Arimidex can successfully knock out genes causing receptors to be insensitive? Pharmacology of Arimidex is very well known, there is absolutely NOTHING in literature that shows this medication can do this even at extremely high breast cancer dosages with extremely long durations. If any med could theorhetically do this, and its a lousy theory, wouldn't it be Nolvadex that actually has play with the receptor itself?</p><p></p><p>If Arimidex could cause estrogen receptor knockout, scientists would not have to use the knockout method aka genetically modified mice DNA to cause successful generations of estrogen insensitivity, they could just feed the mice arimidex, problem is nothing in literature shows Arimidex does this, that is why they don't.</p></blockquote><p></p>
[QUOTE="Vitamin_C, post: 85157, member: 12373"] Do you have labs to indicate you have electrolyte imbalances? DEXA scan to show bone loss or any other lab to show muscle wasting? Do you have labs showing elevated lipids and cholesterol? Elevated prolactin? High CRP? High BP? What do your labs look like pertaining to cortisol and thyroid? Don't just say "they are good".....I'd like to see some actual numbers if you could upload them to the site I'd appreciate it. As a researcher and future practitioner, I am fascinated by these claims but I just see a severe lack of evidence for it. There are women on 1mg of Arimidex a day for sometimes up to 7-10 years, thousands and thousands of women and there is absolutely no data they become insensitive to estrogen via Arimidex. Estrogen is needed for a wide variety of physiological processes in men, from keeping cholesterol in check, prolactin/dopamine etc. I would like to see if you had any lab abnormalities. Like I told nurselyfe, have your endo script you estrogen patches and if you have no high E2 symptoms you can be the first case of this happening in medical history. I am still not understanding the mechanism behind less vascularity and holding water with minuscule amounts of estrogen. Are you having libido/erection problems? Can you show pharmacologically that Arimidex can successfully knock out genes causing receptors to be insensitive? Pharmacology of Arimidex is very well known, there is absolutely NOTHING in literature that shows this medication can do this even at extremely high breast cancer dosages with extremely long durations. If any med could theorhetically do this, and its a lousy theory, wouldn't it be Nolvadex that actually has play with the receptor itself? If Arimidex could cause estrogen receptor knockout, scientists would not have to use the knockout method aka genetically modified mice DNA to cause successful generations of estrogen insensitivity, they could just feed the mice arimidex, problem is nothing in literature shows Arimidex does this, that is why they don't. [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
I accidentally took too much Arimidex...did I screw myself!?
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