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HCG and elevated LH in Alzheimer patients
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<blockquote data-quote="Cataceous" data-source="post: 269494" data-attributes="member: 38109"><p>I have not come across a detailed explanation of the mechanics of HPTA suppression. I speculate that it involves a response to average androgen and estrogen levels over some nebulous time interval. We see that suppression is mostly avoided with short-acting testosterone like Natesto, in spite of high serum peaks. But the half-life of testosterone propionate is relatively long compared to that of Natesto, so I expect it would take quite low doses and/or long breaks to maintain significant HPTA activity while on it. As you suggest, a test of LH might give an idea of what's going on.</p></blockquote><p></p>
[QUOTE="Cataceous, post: 269494, member: 38109"] I have not come across a detailed explanation of the mechanics of HPTA suppression. I speculate that it involves a response to average androgen and estrogen levels over some nebulous time interval. We see that suppression is mostly avoided with short-acting testosterone like Natesto, in spite of high serum peaks. But the half-life of testosterone propionate is relatively long compared to that of Natesto, so I expect it would take quite low doses and/or long breaks to maintain significant HPTA activity while on it. As you suggest, a test of LH might give an idea of what's going on. [/QUOTE]
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HCG and elevated LH in Alzheimer patients
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