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Testosterone Replacement, Low T, HCG, & Beyond
Clomid for PCT, fertility or low T
Clomid only for secondary hypogonadism
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<blockquote data-quote="johndoesmith" data-source="post: 43557" data-attributes="member: 13404"><p>Thank you for the reply.</p><p></p><p>I am just curious how this all works. I understand clomid acts as an estrogen antagonist, and I guess the antagonist effect must be strong. I just cannot wrap my head around how an antagonist can be stronger than NO stimulation from agonists. </p><p></p><p>As in if there's no agonist activity(low estradiol levels for example) how can you have less than no activity, if all an antagonist does is block the agonist(estradiol) from binding to the receptor. In my head it's like sealing shut a door that no one ever uses, how can you have less than zero people going through the door? Or am I just misunderstanding advanced endocrinology?</p></blockquote><p></p>
[QUOTE="johndoesmith, post: 43557, member: 13404"] Thank you for the reply. I am just curious how this all works. I understand clomid acts as an estrogen antagonist, and I guess the antagonist effect must be strong. I just cannot wrap my head around how an antagonist can be stronger than NO stimulation from agonists. As in if there's no agonist activity(low estradiol levels for example) how can you have less than no activity, if all an antagonist does is block the agonist(estradiol) from binding to the receptor. In my head it's like sealing shut a door that no one ever uses, how can you have less than zero people going through the door? Or am I just misunderstanding advanced endocrinology? [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Clomid for PCT, fertility or low T
Clomid only for secondary hypogonadism
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