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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
Can estrogen crash cause desensitization/knock out of the estrogen receptor - lets discuss!
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<blockquote data-quote="nurselyfe" data-source="post: 85827" data-attributes="member: 16355"><p>Ive seen the harvard study. There are plenty of men without ED and anxiety/depression before Finasteride who got it after using it. Like myself, I didn't even know what true anxiety was. Ive had some anxiety over school or girlfriends before - completely normal reactions to life, but not chronic anxiety that can absolutely cripple your mind. Never felt depression until E2 was crashed. By far the worst feeling in the world. While I was crashed I had my Mental Health Clinical rotations and I couldn't have sympathized for these people more. It made me hate that clinical because I knew exactly how they felt. </p><p>This is disturbing that practioners can completely disregard a patients symptoms, and blame their issues on psychiatric problems. As a practioner I would never tell someone who was clearly healthy before that they are manifesting symptoms psychosomatically (albeit they took a drug that causes severe side effects like Propecia), I have strong feelings for doctors like that and don't believe they should be practicing any kind of medicine. </p><p></p><p>I don't do "bro science."I hate anything that is not backed up by literature. But, here I am making an exception because it's real and tangible and no one has or can propose a mechanism yet. I'm speculating two things. </p><p></p><p>1. You have to be lower than 5pg/mL - closer to 0. For a certain period of time using inhibitors, that causes a desensitization of the receptor. </p><p></p><p>2 You have to be extremely low, not necessarily zero, but have extremely quick rebounds in E2 that go much higher in a short period of time, causing negative feedback on receptor signaling. I strongly believe that corepressor proteins have something to do with this. </p><p></p><p>I have NO literature to back this up. But I have 1000's of PFS stories and myself a few others that have on going symptoms. Not just one or two symptoms, but MANY, real, and visible symptoms. </p><p></p><p>I really do not know how much thorough I can be. I told you exactly what kind of person I was before and what I was physically before. I told you every single symtom now that are visible to the eye. I told you things that make me permanently worse when I used to use them for their desired effect. I'm providing NEW bloodwork. Why would I waste my time on here trying to explain myself if nothing was wrong with an already busy life.</p></blockquote><p></p>
[QUOTE="nurselyfe, post: 85827, member: 16355"] Ive seen the harvard study. There are plenty of men without ED and anxiety/depression before Finasteride who got it after using it. Like myself, I didn't even know what true anxiety was. Ive had some anxiety over school or girlfriends before - completely normal reactions to life, but not chronic anxiety that can absolutely cripple your mind. Never felt depression until E2 was crashed. By far the worst feeling in the world. While I was crashed I had my Mental Health Clinical rotations and I couldn't have sympathized for these people more. It made me hate that clinical because I knew exactly how they felt. This is disturbing that practioners can completely disregard a patients symptoms, and blame their issues on psychiatric problems. As a practioner I would never tell someone who was clearly healthy before that they are manifesting symptoms psychosomatically (albeit they took a drug that causes severe side effects like Propecia), I have strong feelings for doctors like that and don't believe they should be practicing any kind of medicine. I don't do "bro science."I hate anything that is not backed up by literature. But, here I am making an exception because it's real and tangible and no one has or can propose a mechanism yet. I'm speculating two things. 1. You have to be lower than 5pg/mL - closer to 0. For a certain period of time using inhibitors, that causes a desensitization of the receptor. 2 You have to be extremely low, not necessarily zero, but have extremely quick rebounds in E2 that go much higher in a short period of time, causing negative feedback on receptor signaling. I strongly believe that corepressor proteins have something to do with this. I have NO literature to back this up. But I have 1000's of PFS stories and myself a few others that have on going symptoms. Not just one or two symptoms, but MANY, real, and visible symptoms. I really do not know how much thorough I can be. I told you exactly what kind of person I was before and what I was physically before. I told you every single symtom now that are visible to the eye. I told you things that make me permanently worse when I used to use them for their desired effect. I'm providing NEW bloodwork. Why would I waste my time on here trying to explain myself if nothing was wrong with an already busy life. [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
Can estrogen crash cause desensitization/knock out of the estrogen receptor - lets discuss!
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