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Not so sure it was using AAS when you were a teen that caused any problems.  Perhaps it is that you ceased to exercise.  Sorry you are having problem with doctors.  Science has not proven that steroids cause left ventricle hypertrophy and instead have proven that it is an adaptation to exercise.  Your HR being lower and stroke volume increasing are also both adaptations to exercise and a sign that your heart is healthy, see below.  Don't know much about UK medicine, but I would fire the cardiologist and get another one.  "Might be" as a diagnose in the field of cardiology is not really acceptable.

Cardiovascular Adaptations to Exercise

Aerobic fitness, anaerobic fitness, and muscular endurance training place larger demands on the heart than any other type of training.  Over time these demands result in adaptations to the cardiovascular system such as:

Physiological Adaptation
Heart Size	The muscular walls of the heart increase in thickness, particularly in the left ventricle, providing a more powerful contraction. The left ventricles internal dimensions increase as a result of increased ventricular filling.
Stroke Volume (SV)	The increase in size of the heart enables the left ventricle to stretch more and thus fill with more blood.  The increase in muscle wall thickness also increases the contractility resulting in increased stroke volume at rest and during exercise, increasing blood supply to the body.
Resting Heart Rate (RHR)	As cardiac output at rest remains constant the increase in stroke volume is accompanied by a corresponding decrease in heart rate.
Cardiac Output (Q)	Cardiac output increases significantly during maximal exercise effort due to the increase in SV.  This results in greater oxygen supply, waste removal and hence improved endurance performance.
Blood Pressure (BP)	People with blood pressure in the ‘normal’ ranges experience little change in BP at rest or with exercise; however hypertensive people find that their BP’s reduce towards normal as they do more exercise.  This is due to a reduction in total peripheral resistance within the artery, and improved condition and elasticity of the smooth muscle in the blood vessel walls.

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