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Testosterone Replacement, Low T, HCG, & Beyond
When Testosterone Is Not Enough
Botox Penile Injections for Erectile Dysfunction
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<blockquote data-quote="madman" data-source="post: 255863" data-attributes="member: 13851"><p><em>*<strong>Multiple organ systems</strong> play a role in achieving a rigid erection, including the cardiovascular, central nervous, peripheral nervous, endocrine, and psychological systems</em></p><p><em></em></p><p><em>*<em><strong>NO</strong> plays a role as a local intrapenile neurotransmitter that facilitates the relaxation of intracavernosal smooth muscle, allowing for increased blood flow into the corpora cavernosa</em></em></p><p><em><em></em></em></p><p><em><em>*<em>The <strong>synergy</strong> of increased arterial inflow and decreased venous outflow allows a man to attain and maintain a firm erection</em></em></em></p><p><em><em><em></em></em></em></p><p><em><em><em>*<em><strong>Cyclic GMP</strong> is needed to maintain nitric oxide-induced dilation of the intracavernosal smooth muscle tissue</em></em></em></em></p><p><em></em></p><p><em></em></p><p><em></em></p><p><em><strong>*The hypothesis behind BoNT A's potential erectogenic effect lies in the idea of an <u>existing balance between sympathetic and parasympathetic influences</u> <u>that maintain flaccidity and rigidity in the penile tissue</u>, respectively. It is believed that <u>BoNT A inhibits the exocytosis of norepinephrine synaptic vesicles at the postganglionic sympathetic neurons in the erectile tissue</u>. <u>The blunting of the sympathetic tone allows NO, which diffuses freely, to be the dominant neurotransmitter within the erectile tissue, thus increasing the overall parasympathetic tone</u></strong></em></p><p></p><p></p><p></p><p></p><p>[URL unfurl="true"]https://www.excelmale.com/forum/threads/botox-for-erectile-dysfunction.25779/[/URL]</p></blockquote><p></p>
[QUOTE="madman, post: 255863, member: 13851"] [I]*[B]Multiple organ systems[/B] play a role in achieving a rigid erection, including the cardiovascular, central nervous, peripheral nervous, endocrine, and psychological systems *[I][B]NO[/B] plays a role as a local intrapenile neurotransmitter that facilitates the relaxation of intracavernosal smooth muscle, allowing for increased blood flow into the corpora cavernosa *[I]The [B]synergy[/B] of increased arterial inflow and decreased venous outflow allows a man to attain and maintain a firm erection *[I][B]Cyclic GMP[/B] is needed to maintain nitric oxide-induced dilation of the intracavernosal smooth muscle tissue[/I][/I][/I] [B]*The hypothesis behind BoNT A's potential erectogenic effect lies in the idea of an [U]existing balance between sympathetic and parasympathetic influences[/U] [U]that maintain flaccidity and rigidity in the penile tissue[/U], respectively. It is believed that [U]BoNT A inhibits the exocytosis of norepinephrine synaptic vesicles at the postganglionic sympathetic neurons in the erectile tissue[/U]. [U]The blunting of the sympathetic tone allows NO, which diffuses freely, to be the dominant neurotransmitter within the erectile tissue, thus increasing the overall parasympathetic tone[/U][/B][/I] [URL unfurl="true"]https://www.excelmale.com/forum/threads/botox-for-erectile-dysfunction.25779/[/URL] [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
When Testosterone Is Not Enough
Botox Penile Injections for Erectile Dysfunction
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