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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone and Men's Health Articles
Assessment of testicular function in boys and adolescents
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<blockquote data-quote="madman" data-source="post: 267400" data-attributes="member: 13851"><p><strong>FIGURE 2 <u>Ontogeny of the hypothalamic‐pituitary‐testicular axis, and the clinical impact of hypogonadism on clinical presentation</u>. The tests differentiate in the first trimester of fetal life, independently of pituitary gonadotropins. Androgens and AMH drive male genital differentiation; in their absence, female differentiation of the genitalia occurs. Hypogonadism in this period leads to ambiguous or female genitalia in XY individuals. In the second and third trimesters, androgens stimulate testicular descent and penile enlargement. Hypogonadism, either primary or central, results in micropenis, cryptorchidism, and/or microorchidism. In newborns, gonadotrophin and steroid secretion are active. Hypogonadism results in reduced penile enlargement. During childhood gonadotrophins and testosterone are low or undetectable. Hypogonadism established in this period does not result in clinically evident signs. During puberty, the gonadal axis is reactivated and results in the development of secondary sex characteristics. Hypogonadism results in absent or incomplete pubertal development.</strong> Reprinted, with permission, from Grinspon et al.4 © 2019 Elsevier Limited. [Color figure can be viewed at wileyonlinelibrary.com]</p><p>[ATTACH=full]38115[/ATTACH]</p></blockquote><p></p>
[QUOTE="madman, post: 267400, member: 13851"] [B]FIGURE 2 [U]Ontogeny of the hypothalamic‐pituitary‐testicular axis, and the clinical impact of hypogonadism on clinical presentation[/U]. The tests differentiate in the first trimester of fetal life, independently of pituitary gonadotropins. Androgens and AMH drive male genital differentiation; in their absence, female differentiation of the genitalia occurs. Hypogonadism in this period leads to ambiguous or female genitalia in XY individuals. In the second and third trimesters, androgens stimulate testicular descent and penile enlargement. Hypogonadism, either primary or central, results in micropenis, cryptorchidism, and/or microorchidism. In newborns, gonadotrophin and steroid secretion are active. Hypogonadism results in reduced penile enlargement. During childhood gonadotrophins and testosterone are low or undetectable. Hypogonadism established in this period does not result in clinically evident signs. During puberty, the gonadal axis is reactivated and results in the development of secondary sex characteristics. Hypogonadism results in absent or incomplete pubertal development.[/B] Reprinted, with permission, from Grinspon et al.4 © 2019 Elsevier Limited. [Color figure can be viewed at wileyonlinelibrary.com] [ATTACH type="full"]38115[/ATTACH] [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone and Men's Health Articles
Assessment of testicular function in boys and adolescents
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