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Testosterone Replacement, Low T, HCG, & Beyond
Blood Test Discussion
Aspirin before CBC test to affect Hematocrit
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<blockquote data-quote="madman" data-source="post: 148304" data-attributes="member: 13851"><p>Aspirin will have an effect on platelets not hematocrit.</p><p></p><p>The only way aspirin would lower ones hematocrit level is when it causes gastrointestinal bleeding.</p><p></p><p></p><p></p><p></p><p><strong><span style="color: rgb(184, 49, 47)">Aspirin </span></strong>is also used as an <span style="color: rgb(184, 49, 47)"><strong>antiplatelet/platelet aggregation inhibitor (</strong></span><span style="color: rgb(44, 130, 201)"><strong>to keep your blood from sticking together</strong></span><span style="color: rgb(184, 49, 47)"><strong>)</strong></span><span style="color: rgb(0, 0, 0)">.</span></p><p></p><p>Regarding water intake hydration/electrolytes is critical and will impact hematocrit levels.</p><p></p><p>Forget about just hydrating the day of lab work.....you should concentrate on fluid intake/electrolytes days before and make sure to add some sodium (himalayan, sea salt) to your diet.</p><p></p><p></p><p></p><p></p><p></p><p><strong>Aspirin</strong></p><p><span style="color: rgb(184, 49, 47)">Mechanism of action:</span> Aspirin (acetylsalicylic acid) irreversibly inhibits prostaglandin H synthase (cyclooxygenase-1) in platelets and megakaryocytes, and thereby<span style="color: rgb(184, 49, 47)"> <strong>blocks</strong></span><span style="color: rgb(0, 0, 0)"><strong> the </strong></span><span style="color: rgb(184, 49, 47)"><strong>formation of thromboxane A2</strong></span><span style="color: rgb(0, 0, 0)"><strong> (</strong></span><span style="color: rgb(184, 49, 47)"><strong>TXA2</strong></span><span style="color: rgb(0, 0, 0)"><strong>; a </strong></span><span style="color: rgb(44, 130, 201)"><strong>potent vasoconstrictor</strong></span><span style="color: rgb(0, 0, 0)"><strong> and </strong></span><span style="color: rgb(44, 130, 201)"><strong>platelet aggregant</strong></span><span style="color: rgb(0, 0, 0)"><strong>)</strong></span>. <strong>It is only the <span style="color: rgb(184, 49, 47)">parent form, acetylsalicylic acid</span>, which has any<span style="color: rgb(184, 49, 47)"> significant effect </span>on <span style="color: rgb(184, 49, 47)">platelet function</span></strong>. <strong>Because <span style="color: rgb(184, 49, 47)">platelets</span> are <span style="color: rgb(184, 49, 47)">unable to regenerate</span> <span style="color: rgb(44, 130, 201)">cyclooxygenase</span>, the <span style="color: rgb(44, 130, 201)">immediate antithrombotic effect</span> of aspirin remains for the <span style="color: rgb(184, 49, 47)">lifespan of the platelet (8–10 days)</span></strong>. As, after stopping aspirin therapy, normal haemostasis may be regained when about 20% of platelets have normal cyclooxygenase activity, daily aspirin intake is recommended.</p></blockquote><p></p>
[QUOTE="madman, post: 148304, member: 13851"] Aspirin will have an effect on platelets not hematocrit. The only way aspirin would lower ones hematocrit level is when it causes gastrointestinal bleeding. [B][COLOR=rgb(184, 49, 47)]Aspirin [/COLOR][/B]is also used as an [COLOR=rgb(184, 49, 47)][B]antiplatelet/platelet aggregation inhibitor ([/B][/COLOR][COLOR=rgb(44, 130, 201)][B]to keep your blood from sticking together[/B][/COLOR][COLOR=rgb(184, 49, 47)][B])[/B][/COLOR][COLOR=rgb(0, 0, 0)].[/COLOR] Regarding water intake hydration/electrolytes is critical and will impact hematocrit levels. Forget about just hydrating the day of lab work.....you should concentrate on fluid intake/electrolytes days before and make sure to add some sodium (himalayan, sea salt) to your diet. [B]Aspirin[/B] [COLOR=rgb(184, 49, 47)]Mechanism of action:[/COLOR] Aspirin (acetylsalicylic acid) irreversibly inhibits prostaglandin H synthase (cyclooxygenase-1) in platelets and megakaryocytes, and thereby[COLOR=rgb(184, 49, 47)] [B]blocks[/B][/COLOR][COLOR=rgb(0, 0, 0)][B] the [/B][/COLOR][COLOR=rgb(184, 49, 47)][B]formation of thromboxane A2[/B][/COLOR][COLOR=rgb(0, 0, 0)][B] ([/B][/COLOR][COLOR=rgb(184, 49, 47)][B]TXA2[/B][/COLOR][COLOR=rgb(0, 0, 0)][B]; a [/B][/COLOR][COLOR=rgb(44, 130, 201)][B]potent vasoconstrictor[/B][/COLOR][COLOR=rgb(0, 0, 0)][B] and [/B][/COLOR][COLOR=rgb(44, 130, 201)][B]platelet aggregant[/B][/COLOR][COLOR=rgb(0, 0, 0)][B])[/B][/COLOR]. [B]It is only the [COLOR=rgb(184, 49, 47)]parent form, acetylsalicylic acid[/COLOR], which has any[COLOR=rgb(184, 49, 47)] significant effect [/COLOR]on [COLOR=rgb(184, 49, 47)]platelet function[/COLOR][/B]. [B]Because [COLOR=rgb(184, 49, 47)]platelets[/COLOR] are [COLOR=rgb(184, 49, 47)]unable to regenerate[/COLOR] [COLOR=rgb(44, 130, 201)]cyclooxygenase[/COLOR], the [COLOR=rgb(44, 130, 201)]immediate antithrombotic effect[/COLOR] of aspirin remains for the [COLOR=rgb(184, 49, 47)]lifespan of the platelet (8–10 days)[/COLOR][/B]. As, after stopping aspirin therapy, normal haemostasis may be regained when about 20% of platelets have normal cyclooxygenase activity, daily aspirin intake is recommended. [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Blood Test Discussion
Aspirin before CBC test to affect Hematocrit
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