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<blockquote data-quote="Rand McClain DO" data-source="post: 88229" data-attributes="member: 90"><p>We, the medical community, are gaining more information regarding prostate cancer and testosterone use geometrically rapidly since Dr. Morgenthaler pointed out the flaws in the study that was the basis for which 70 years of dogma re testosterone causing prostate cancer was made. </p><p>We now have evidence that low testosterone is correlated with prostate cancer and that some estrogens activate the genes (which all men carry) for prostate cancer.</p><p>Recently, a small study (n=15) showed that cyclic use of testosterone can reverse or cure prostate cancer. </p><p>I’ll share that I was diagnosed with prostate cancer over two years ago and have been cancer free for two years after treatment using green tea extract with capsaicin (“Capsol-T”), Metformin, dutasteride and testosterone along with other forms of nutritional, supplemental and lifestyle support.</p><p>Standard of care still uses chemical castration, estrogen, radiation and prostatectomy as first line treatments with variable results and many side effects - many permanent. And, standard of care still uses PSA to screen for prostate cancer despite the proven lack of efficacy and the inventor of the test asking it no longer be used for screening because of the consistent false positives and negatives and frequent unwarranted biopsies that have plenty of unwanted consequences as well.</p><p>A PSA can be used, however, to screen for possible undetected metastases of prostate cancer in cases where the prostate and all prostate cancer has been presumed to be removed.</p><p>I have patients with successfully treated prostate cancer using TRT and none have had recurrence of prostate cancer. I suggest to any patient concerned about cancer to submit to an ONCOblot assay which evaluates for the presence of ENOX-2 proteins (found only in cancer or fetal tissue) and by weight and pH can determine tissue type for at least 26 cancers. Early detection is key and if used properly (ie frequently enough) the ONCOblot test should be used with available treatments to treat successfully most cancers.</p><p>So, if concern exists about extant prostate cancer present after treatment, PSA, ONCOblot, and/or MRI (can detect lesions as small as 3mm) can be used to rule it in or out. DHT has definitely been shown to fuel prostate cancer so I recommend use of a 5-alpha reductase inhibitor if prostate tissue is still present. As previously stated, certain estrogens also activate the genes for prostate cancer which is why I recommend using an aromatase inhibitor to keep estrogens as low as possible while still keeping enough requisite for heart, brain and joint health. The use of DIM or consumption of ample cruciferous vegetables can help prevent conversion of necessary/beneficial estrogens to harmful estrogens.</p><p>I hope this allays any concerns you and others may have about resuming TRT after successful treatment (or as yet unsuccessful treatment) of prostate cancer.</p></blockquote><p></p>
[QUOTE="Rand McClain DO, post: 88229, member: 90"] We, the medical community, are gaining more information regarding prostate cancer and testosterone use geometrically rapidly since Dr. Morgenthaler pointed out the flaws in the study that was the basis for which 70 years of dogma re testosterone causing prostate cancer was made. We now have evidence that low testosterone is correlated with prostate cancer and that some estrogens activate the genes (which all men carry) for prostate cancer. Recently, a small study (n=15) showed that cyclic use of testosterone can reverse or cure prostate cancer. I’ll share that I was diagnosed with prostate cancer over two years ago and have been cancer free for two years after treatment using green tea extract with capsaicin (“Capsol-T”), Metformin, dutasteride and testosterone along with other forms of nutritional, supplemental and lifestyle support. Standard of care still uses chemical castration, estrogen, radiation and prostatectomy as first line treatments with variable results and many side effects - many permanent. And, standard of care still uses PSA to screen for prostate cancer despite the proven lack of efficacy and the inventor of the test asking it no longer be used for screening because of the consistent false positives and negatives and frequent unwarranted biopsies that have plenty of unwanted consequences as well. A PSA can be used, however, to screen for possible undetected metastases of prostate cancer in cases where the prostate and all prostate cancer has been presumed to be removed. I have patients with successfully treated prostate cancer using TRT and none have had recurrence of prostate cancer. I suggest to any patient concerned about cancer to submit to an ONCOblot assay which evaluates for the presence of ENOX-2 proteins (found only in cancer or fetal tissue) and by weight and pH can determine tissue type for at least 26 cancers. Early detection is key and if used properly (ie frequently enough) the ONCOblot test should be used with available treatments to treat successfully most cancers. So, if concern exists about extant prostate cancer present after treatment, PSA, ONCOblot, and/or MRI (can detect lesions as small as 3mm) can be used to rule it in or out. DHT has definitely been shown to fuel prostate cancer so I recommend use of a 5-alpha reductase inhibitor if prostate tissue is still present. As previously stated, certain estrogens also activate the genes for prostate cancer which is why I recommend using an aromatase inhibitor to keep estrogens as low as possible while still keeping enough requisite for heart, brain and joint health. The use of DIM or consumption of ample cruciferous vegetables can help prevent conversion of necessary/beneficial estrogens to harmful estrogens. I hope this allays any concerns you and others may have about resuming TRT after successful treatment (or as yet unsuccessful treatment) of prostate cancer. [/QUOTE]
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