Hi eudes, you are very welcome. I can Skype with you if you like.
Regarding your concern for your lipids, as long as you are not OVERsuppressing your estrogen, you should have no negative effect on your HDL. You can avoid gynecomastia (estrogen related) by keeping your E within normal limits, and keeping it so, will not negatively affect your lipids.
One consideration: if you are 30 years old or older, and you have no signs of male pattern baldness yet, it is unlikely you will develop it and therefore you may have less concern for modulating your DHT downward. Remember that Receding Hairline is also genetic, but not affected by excess DHT, so use of a 5-alpha reductase inhibitor to attempt to treat this would be relatively fruitless.
Taking Raloxifen while taking Arimidex will reduce the efficacy of Arimidex somewhat (about 13% if I remember correctly), and theoretically after about 4 days of Arimidex, you would not benefit from an E receptor blocker like Raloxifen. BUT, I will say that, clinically, I have seen benefit in the treatment of acute onset gynecomastia using tamoxifen, even when using Arimidex to keep E levels optimal, so I would consider sticking with your plan if your gynecomastia is newly developed. While it may shrink completely if new, I find that often what gynecomastia is diminished can be enough to make it no longer a nuisance, but that some breast tissue still remains. When removing gynecomastia, I regularly find, say, a golf ball sized tissue mass once excised, yet upon palpation presurgically, I felt the presence of, say, a marble sized mass.
