ExcelMale
Menu
Home
What's new
Latest activity
Forums
New posts
Search forums
What's new
New posts
Latest activity
Videos
Lab Tests
Doctor Finder
Buy Books
About Us
Men’s Health Coaching
Log in
Register
What's new
Search
Search
Search titles only
By:
New posts
Search forums
Menu
Log in
Register
Navigation
Install the app
Install
More options
Contact us
Close Menu
Forums
Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Basics & Questions
Anyone taking Statins ( Crestor )
JavaScript is disabled. For a better experience, please enable JavaScript in your browser before proceeding.
You are using an out of date browser. It may not display this or other websites correctly.
You should upgrade or use an
alternative browser
.
Reply to thread
Message
<blockquote data-quote="bp" data-source="post: 164108" data-attributes="member: 38377"><p>'familial hypercholesterolemia' is THE genetic mutation which creates a higher level of LDL concentration. People with fH have very high levels of LDL-c. Yet, many with fH suffer no CVD at all,..none, nada. Hence the reality that LDL-c levels have nothing to do with CVD.</p><p></p><p>LDL-p becomes a different issue , and has a small relevance to CVD, and only when severe oxidation is taking place, (which can be negated through high dose vit e )but still, however, this is almost negligible when put next to TG, systemic inflammation and clotting abnormalities.</p><p></p><p>LDL is found to be deposited in the thrombus in the lining of the intima in arterial plaque, yet, the percentage by which the LDL is deposited has no bearing on the level of LDL-c or -p circulating. ( The LDL in the plaque deposits dont correlate to high LDL-c or -P)</p><p></p><p>Hence why heart attacks through infarction is seen with patients with low, medium and high LDL-c and -P numbers</p><p></p><p>The other problem is that the majority of stain medicines have little or no effect on LDL-p (which is only mildly correlated to cvd), and no effect at all on Lp(a) which is a better risk factor in the relms of lipid issues than LDL-p is.</p><p></p><p>The actual reason why statins do have a positive effect on CVD is nothing to do with the ldl-c lowering properties (which are pointless), but everything to do with the mechanism by which they promote anti inflammation in the endothelial lining of the coronary artery wall.</p><p></p><p>Its actually just damn good luck they they (statins) exhibit these properties. Otherwise, theyd be beyond pointless.</p><p></p><p>The same results for FMD and correction of endothelial dysfunction can be created by using ACE inhibitors and folate.</p><p></p><p>If youd like any info on this CVD, feel free to pm me, im up to 328 clinical trials read now in the last 13 years involving CVD, plus can put u in touch with the cardiologists who arent 'statin whisperers'</p></blockquote><p></p>
[QUOTE="bp, post: 164108, member: 38377"] 'familial hypercholesterolemia' is THE genetic mutation which creates a higher level of LDL concentration. People with fH have very high levels of LDL-c. Yet, many with fH suffer no CVD at all,..none, nada. Hence the reality that LDL-c levels have nothing to do with CVD. LDL-p becomes a different issue , and has a small relevance to CVD, and only when severe oxidation is taking place, (which can be negated through high dose vit e )but still, however, this is almost negligible when put next to TG, systemic inflammation and clotting abnormalities. LDL is found to be deposited in the thrombus in the lining of the intima in arterial plaque, yet, the percentage by which the LDL is deposited has no bearing on the level of LDL-c or -p circulating. ( The LDL in the plaque deposits dont correlate to high LDL-c or -P) Hence why heart attacks through infarction is seen with patients with low, medium and high LDL-c and -P numbers The other problem is that the majority of stain medicines have little or no effect on LDL-p (which is only mildly correlated to cvd), and no effect at all on Lp(a) which is a better risk factor in the relms of lipid issues than LDL-p is. The actual reason why statins do have a positive effect on CVD is nothing to do with the ldl-c lowering properties (which are pointless), but everything to do with the mechanism by which they promote anti inflammation in the endothelial lining of the coronary artery wall. Its actually just damn good luck they they (statins) exhibit these properties. Otherwise, theyd be beyond pointless. The same results for FMD and correction of endothelial dysfunction can be created by using ACE inhibitors and folate. If youd like any info on this CVD, feel free to pm me, im up to 328 clinical trials read now in the last 13 years involving CVD, plus can put u in touch with the cardiologists who arent 'statin whisperers' [/QUOTE]
Insert quotes…
Verification
Post reply
Share this page
Facebook
Twitter
Reddit
Pinterest
Tumblr
WhatsApp
Email
Share
Link
Sponsors
Forums
Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Basics & Questions
Anyone taking Statins ( Crestor )
This site uses cookies to help personalise content, tailor your experience and to keep you logged in if you register.
By continuing to use this site, you are consenting to our use of cookies.
Accept
Learn more…
Top