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Thyroid, Pregnenolone, Progesterone, DHEA, etc
Thyroid, DHEA, Pregnenolone, Progesterone, etc
Androgens make hyperthyroid worse?
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<blockquote data-quote="apsjiml" data-source="post: 166467" data-attributes="member: 12831"><p>You cannot be hyperthyroid with a free t3 in the normal range. Reverse t3 is made from t4, so if you want to have less reverse t3 you need less t4. Most people that do not have a conversion issue really do have a reverse t3 at the bottom of the range. Like my brothers was 8,9, 11 same as my dads but mine was 36 or more. So, reverse t3 most will tell you it is non thyroidal illness that causes that. The problem is when we are talking deiodinase (fancy term for enzymes responsible for t4 into the active hormone which is free t3) lower testosterone, lower growth hormone, lower iron all cause issues with thyroid conversion leading to higher reverse t3. There are also various genetic gene defects that cause this issue and a big one is ANY inflammation can cause lack of thyroid conversion, that could be back injury or hashimoto's or other autoimmune disorders , any inflammation of the body like that can cause a issue with thyroid conversion. One of the issues with the current essays is lack of sensitivity because rt3 and t3 look so similiar it can be difficult to tell them apart. I see when people take in less t4 (usually 50mcg or less ) and the rest t3 their free t3 levels go down when they get rt3 down . lc-ms/ms is suppose to be more sensitive but not always available. One last thing, If you have Hashimoto's with elevated antibodies, you should be looking into Low Dose Naltrexone to get those antibodies down. If you have not yet, you should be testing both Hashimoto's antibodies . Ldn actually lowers inflammation too.</p></blockquote><p></p>
[QUOTE="apsjiml, post: 166467, member: 12831"] You cannot be hyperthyroid with a free t3 in the normal range. Reverse t3 is made from t4, so if you want to have less reverse t3 you need less t4. Most people that do not have a conversion issue really do have a reverse t3 at the bottom of the range. Like my brothers was 8,9, 11 same as my dads but mine was 36 or more. So, reverse t3 most will tell you it is non thyroidal illness that causes that. The problem is when we are talking deiodinase (fancy term for enzymes responsible for t4 into the active hormone which is free t3) lower testosterone, lower growth hormone, lower iron all cause issues with thyroid conversion leading to higher reverse t3. There are also various genetic gene defects that cause this issue and a big one is ANY inflammation can cause lack of thyroid conversion, that could be back injury or hashimoto's or other autoimmune disorders , any inflammation of the body like that can cause a issue with thyroid conversion. One of the issues with the current essays is lack of sensitivity because rt3 and t3 look so similiar it can be difficult to tell them apart. I see when people take in less t4 (usually 50mcg or less ) and the rest t3 their free t3 levels go down when they get rt3 down . lc-ms/ms is suppose to be more sensitive but not always available. One last thing, If you have Hashimoto's with elevated antibodies, you should be looking into Low Dose Naltrexone to get those antibodies down. If you have not yet, you should be testing both Hashimoto's antibodies . Ldn actually lowers inflammation too. [/QUOTE]
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Thyroid, Pregnenolone, Progesterone, DHEA, etc
Thyroid, DHEA, Pregnenolone, Progesterone, etc
Androgens make hyperthyroid worse?
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