As far as i remember,Patrick Arnold the chemist had written an article in his website.
He said that even non aromatised AAS (flyoxymesterone in specific),elevate systemic blood pressure,although there is no estrogenic effect.Well,halotestin (stexon brand name) is one of these highly androgenic...
A 21year old male,fully recovered from a 24week AAS cycle,followed by a four week PCT.
Test enanthate,Tren acetate,Equipose,Anastrozole and Cabergoline were the ergogenics.
PCT included hCG & SERMs afterwords.
Rhabdomyolysis,as a result of hypertraining and cocaine use.Transaminemia,as skeletal muscles have common receptors with liver and myocardium.Slight hyperkalemia as a result of striated muscle breakdown (Potassium is the main inrracellular element).Creatinine at higher optimal range,due to toxic...
A 50yo male visited me,in order to evaluate his HPTA.
His major concern was if there is any case of andropause.
However,during the medical history,there were no clinical symptoms of hypogonadism.As we are aware of,symptoms have to be followed by labs in order to diagnose andropause.
It is...
Transaminemia (AST/ALT) and dislipidemia (HDL/LDL/TC),as a result of AAS abuse.
Slight rhabdomyolysis effect as well.
All labs were back to normal,six weeks after.
NAC,LIV52,Milk thisle,Lecithin,Οmega 369,
Phytosterols,Niacin and Krill oil use used on a daily basis plan.
45yo male with primary hypogonadism,undergoes a PCT with HCG & SERMs use.
Libido was improved with overall mental/physical state.
DHEA supplementation seems obligatory.
A typical laboratory finding of andropause
fter four bottles (16ml/16gr)
of Nebido/Aveed (Testosterone Undecaonate)
25mg TU every day
25mg DHEA every day
25mg DHT every day
100iu HCG every day
1mg Anastrozol per week
July 2017/November 2017
This was a case of a patient who had iron deficiency anemia.MCV was considerable high,but H/H were frustrating low,so were iron & ferritin.Thalasemia patients have considerable low MCV and they are obligated to take folic acid for life.The cell membrane of their RBCs is more fragile and...
CVD panel also includes:
Homocysteine and Lipoprotein A
In CBC when MCV is lower than 70, we have thalasemia.This is a non treated anemia,where RBCs appear to be smaller in size.
H/H in these patients never gets above 45%/15 and is usually its close to 40%/13.5
ALP & GGT are cholestatic...
Low SHBG is something you notice under AAS cycles.Also DHT can lower SHBG,as well as synthetic derivatives (oxandrolone,stanozolol,methenolone) and synthetic DHT as well (mesterolone,drostanolone).
As DHT bonds tight to SHBG,it permits more FT to circulate.This is something good apparently,for...
In case of an elevated CPK,doctors also check out fragment CK-MB of myocardium.Usually,its elevated under AMI,along with troponin protein,ALT,AST,LDH.All these biochemical measurements are part of a damaged heart muscle.
Another case is rhabdomyolysis effect,under vigorous physical activity.Also...
Men must donate,either they are on HRT,or not.Androgens stimulate EPO anyway.This is why men have higher hemoglobin than women.
Too often(<120days),may boost red bone marrow and EPO production,indirectly.It can also tank your serum iron and iron stores (ferritin protein).
https://www.amazon.com/PEDS-use-sports-Physicians-Perspective-ebook/dp/B073DCXG46/ref=sr_1_3?ie=UTF8&qid=1498653891&sr=8-3&keywords
E-book is already available
I am glad to announce,that my next project will be released on Amazon,by this summer.
It is the expanded version of my previous book in Greek,focusing on medical prevention rules,regarding PEDs use and principles of PCT/HRT,as a result to AAS abuse.
The text encloses more than 35,000...
http://gtoul.com/pct-post-cycle-therapy/
http://gtoul.com/suppression-of-the-pituitary-hypothalamic-testicular-axis-as-a-result-of-aas-abuse/
These links will lighten up
In old men,as cachexia proceeds,fat tissue accumulates.Estradiol peaks and that lowers FT,because SHBG elevates.
Therefore muscle mass can lower SHBG indirectly?
Heart muscle possesses also striated fibers.
Therefore myocardium reacts both in resistance training and AAS,since androgen receptors are located in several tissues.
LVH is the result of high intensity training.
Either CV,or resistance.
In order to compensate the wall thickening,low pace cardio...
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