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Statin Drugs Markedly Inhibit Testosterone Production
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<blockquote data-quote="Nelson Vergel" data-source="post: 57860" data-attributes="member: 3"><p>Wow, SoCal...great paper! Scary indeed.</p><p></p><p><span style="color: #333333">Figure 4. </span>Comparison of skeletal muscle properties in the leg between statin users and statin non-users. As compared with non-users, statin users exhibited higher systolic blood pressure and elevated glycated hemoglobin (HbA1c) level. Levels of CoQ10, anti-peroxidative enzymes, uncoupling protein (UCP), Complex IV, and myosin were lower, oxidative phosphorylation ability was lower and glucose level in glucose tolerance test was higher</p><p></p><p><strong>[ATTACH]2911[/ATTACH]</strong></p><p><strong></strong></p><p><strong></strong></p><p><strong>"</strong><span style="color: #333333">Pharmacological evidence and clinical trial results support the interpretation that statins stimulate atherogenesis by suppressing vitamin K2 synthesis and thereby enhancing artery calcification. Statins cause heart failure by depleting the myocardium of CoQ10, & heme A' and selenoproteins, thereby impairing mitochondrial ATP production. In summary, statins are not only ineffective in preventing CHD events but instead are capable of increasing CHD and heart failure. Physicians who are involved in prescribing cholesterol-lowering medications cannot ignore the moral responsibility of informed consent'. Patients must be informed of all statin adverse effects, including the ability to cause CHD and heart failure, onset of diabetes mellitus, carcinogenicity, teratogenicity and central and peripheral nervous disorders besides the well-known rhabdomyolysis and hepatic injury. Most of these adverse effects of statins become apparent after 6 or more years of statin therapy. Chronic administration could ultimately lead to these statin adverse effects as pharmaceutical and biochemical research has now demonstrated."</span></p></blockquote><p></p>
[QUOTE="Nelson Vergel, post: 57860, member: 3"] Wow, SoCal...great paper! Scary indeed. [COLOR=#333333]Figure 4. [/COLOR]Comparison of skeletal muscle properties in the leg between statin users and statin non-users. As compared with non-users, statin users exhibited higher systolic blood pressure and elevated glycated hemoglobin (HbA1c) level. Levels of CoQ10, anti-peroxidative enzymes, uncoupling protein (UCP), Complex IV, and myosin were lower, oxidative phosphorylation ability was lower and glucose level in glucose tolerance test was higher [B][ATTACH alt="statins negative effects.jpg"]2911[/ATTACH] "[/B][COLOR=#333333]Pharmacological evidence and clinical trial results support the interpretation that statins stimulate atherogenesis by suppressing vitamin K2 synthesis and thereby enhancing artery calcification. Statins cause heart failure by depleting the myocardium of CoQ10, & heme A' and selenoproteins, thereby impairing mitochondrial ATP production. In summary, statins are not only ineffective in preventing CHD events but instead are capable of increasing CHD and heart failure. Physicians who are involved in prescribing cholesterol-lowering medications cannot ignore the moral responsibility of informed consent'. Patients must be informed of all statin adverse effects, including the ability to cause CHD and heart failure, onset of diabetes mellitus, carcinogenicity, teratogenicity and central and peripheral nervous disorders besides the well-known rhabdomyolysis and hepatic injury. Most of these adverse effects of statins become apparent after 6 or more years of statin therapy. Chronic administration could ultimately lead to these statin adverse effects as pharmaceutical and biochemical research has now demonstrated."[/COLOR] [/QUOTE]
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Statin Drugs Markedly Inhibit Testosterone Production
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