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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
Estradiol in Men: Libido and Brain Function
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<blockquote data-quote="Nelson Vergel" data-source="post: 197903" data-attributes="member: 3"><p>"</p><p><strong><em><u>As noted above, the serum concentration of E-2 plays a dominant role in male sexual health</u>. Low levels of aromatase, which result in extremely low E-2 concentrations, may, nevertheless, be important in male sexual performance without affecting sexual orientation and gender identity (Gomuła, 2006, Gomuła, 2007). In patients with congenital absence of aromatase only the low-dose E-2 substitution results in significant changes in sexual behavior. <u>Estradiol administration leads to increased erotic fantasies, masturbation, or sexual activity (Carani, et al., 1999)</u>.</em></strong></p><p><strong><em></em></strong></p><p><strong><em><u>My own research clearly shows that E-2 is required to maintain sexual functions in adult men (Gomuła, 2007)</u>. The manifestations thereof have been observed after prolonged hormonal therapy. Androgen deficiency patients had their testosterone levels increased for therapeutic purposes. During the therapy, a parallel increase occurred in serum E-2 concentrations, as the effect of the all-natural aromatase. Some patients had such high E-2 levels that they exceeded the normal physiological range. In order to reduce E-2 concentration, my patients received preparations blocking aromatase activity. <u>As a result of this therapy, men characterized by high concentrations of testosterone (falling in the upper limits of normal), whose E-2 was detected at very low levels, had a total loss of libido</u>. <u>At the same time, those men suffered from erectile dysfunction, which could even lead to the inability to initiate or maintain an erection</u>. <u>Stopping the drug which blocked aromatase resolved the symptoms and resulted in a rapid return of high concentrations of E-2. Some authors report that in the activation of male sexual behavior the brain level conversion of testosterone to estradiol is of major importance and that testosterone's effects are not in themselves so important (Balthazar & Ball, 1998)</u>. Testosterone has a significant effect on the smooth muscle in the corpora cavernosa. Androgens may significantly affect the ultrastructure of the corpora cavernosa and these changes are responsible for erectile dysfunction (Traish & Kim, 2005)."</em></strong></p><p><strong><em></em></strong></p><p><strong><em>[URL unfurl="true"]https://www.excelmale.com/forum/threads/late-onset-hypogonadism-update.23078/[/URL]</em></strong></p></blockquote><p></p>
[QUOTE="Nelson Vergel, post: 197903, member: 3"] " [B][I][U]As noted above, the serum concentration of E-2 plays a dominant role in male sexual health[/U]. Low levels of aromatase, which result in extremely low E-2 concentrations, may, nevertheless, be important in male sexual performance without affecting sexual orientation and gender identity (Gomuła, 2006, Gomuła, 2007). In patients with congenital absence of aromatase only the low-dose E-2 substitution results in significant changes in sexual behavior. [U]Estradiol administration leads to increased erotic fantasies, masturbation, or sexual activity (Carani, et al., 1999)[/U]. [U]My own research clearly shows that E-2 is required to maintain sexual functions in adult men (Gomuła, 2007)[/U]. The manifestations thereof have been observed after prolonged hormonal therapy. Androgen deficiency patients had their testosterone levels increased for therapeutic purposes. During the therapy, a parallel increase occurred in serum E-2 concentrations, as the effect of the all-natural aromatase. Some patients had such high E-2 levels that they exceeded the normal physiological range. In order to reduce E-2 concentration, my patients received preparations blocking aromatase activity. [U]As a result of this therapy, men characterized by high concentrations of testosterone (falling in the upper limits of normal), whose E-2 was detected at very low levels, had a total loss of libido[/U]. [U]At the same time, those men suffered from erectile dysfunction, which could even lead to the inability to initiate or maintain an erection[/U]. [U]Stopping the drug which blocked aromatase resolved the symptoms and resulted in a rapid return of high concentrations of E-2. Some authors report that in the activation of male sexual behavior the brain level conversion of testosterone to estradiol is of major importance and that testosterone's effects are not in themselves so important (Balthazar & Ball, 1998)[/U]. Testosterone has a significant effect on the smooth muscle in the corpora cavernosa. Androgens may significantly affect the ultrastructure of the corpora cavernosa and these changes are responsible for erectile dysfunction (Traish & Kim, 2005)." [URL unfurl="true"]https://www.excelmale.com/forum/threads/late-onset-hypogonadism-update.23078/[/URL][/I][/B] [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
Estradiol in Men: Libido and Brain Function
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