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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
hCG and Vision
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<blockquote data-quote="Nelson Vergel" data-source="post: 9724" data-attributes="member: 3"><p>The best part of this paper is the introduction that describes the effects of HCG on the CNS and other tissues. I have been trying to find good references on this, so thanks for this great find.</p><p></p><p>"The &#945; subunit of hCG is identical to &#945; subunits of other human glycoproteins: luteinizing hormone (LH), follicle stimulating hormone (FSH), and the thyroid stimulating hormone (TSH); the &#946; subunit therefore provides hormonal specificity <a href="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Bellisario1" target="_blank">[1]</a>. In the past decades, aside from its role in pregnancy, several reports showed that hCG together with LH elicit multiple effects in the central nervous system <a href="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Lei1" target="_blank">[2]</a>. Many of the behavioral changes induced by hCG injection in rats parallel those observed in pregnant women <a href="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Bowen1" target="_blank">[3]</a> and some of these behavioral effects are correlated with changes of eicosanoid metabolism induced by LH and hCG in the brain <a href="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Toth1" target="_blank">[4]</a>. Administration of gonadotropins can induce appetite loss, facilitation of extinction of the conditioned avoidance response, decreased exploratory activity, and decreased electrical activity of the brain <a href="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Kawakami1" target="_blank">[5]</a>&#8211;<a href="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Lukcs1" target="_blank">[8]</a>. In addition, trophic effects of hCG have been identified during development, neuroregenerative processes and tumorigenesis in CNS. Culturing fetal brain neurons, in the presence of highly purified hCG, resulted in a dose-dependent increase of survival and of neurite outgrowth <a href="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-AlHader1" target="_blank">[9]</a>. Treatment of rats with hCG after a complete transection of the spinal cord induced the presence of nerve fibers in the bridging tissue suggesting that hCG might be useful in functional recovery for patients with paraplegia<a href="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Patil1" target="_blank">[10]</a>. "</p></blockquote><p></p>
[QUOTE="Nelson Vergel, post: 9724, member: 3"] The best part of this paper is the introduction that describes the effects of HCG on the CNS and other tissues. I have been trying to find good references on this, so thanks for this great find. "The α subunit of hCG is identical to α subunits of other human glycoproteins: luteinizing hormone (LH), follicle stimulating hormone (FSH), and the thyroid stimulating hormone (TSH); the β subunit therefore provides hormonal specificity [URL="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Bellisario1"][1][/URL]. In the past decades, aside from its role in pregnancy, several reports showed that hCG together with LH elicit multiple effects in the central nervous system [URL="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Lei1"][2][/URL]. Many of the behavioral changes induced by hCG injection in rats parallel those observed in pregnant women [URL="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Bowen1"][3][/URL] and some of these behavioral effects are correlated with changes of eicosanoid metabolism induced by LH and hCG in the brain [URL="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Toth1"][4][/URL]. Administration of gonadotropins can induce appetite loss, facilitation of extinction of the conditioned avoidance response, decreased exploratory activity, and decreased electrical activity of the brain [URL="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Kawakami1"][5][/URL]–[URL="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Lukcs1"][8][/URL]. In addition, trophic effects of hCG have been identified during development, neuroregenerative processes and tumorigenesis in CNS. Culturing fetal brain neurons, in the presence of highly purified hCG, resulted in a dose-dependent increase of survival and of neurite outgrowth [URL="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-AlHader1"][9][/URL]. Treatment of rats with hCG after a complete transection of the spinal cord induced the presence of nerve fibers in the bridging tissue suggesting that hCG might be useful in functional recovery for patients with paraplegia[URL="http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0052567#pone.0052567-Patil1"][10][/URL]. " [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
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