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Testosterone Replacement, Low T, HCG, & Beyond
When Testosterone Is Not Enough
Sexual Dysfunction in Men After a Heart Attack
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<blockquote data-quote="Nelson Vergel" data-source="post: 10596" data-attributes="member: 3"><p>OBJECTIVES: The aim of the study is to assess the frequency of sexual dysfunction in men after myocardial infarction (MI).</p><p></p><p>METHODS: 62 men were asked to fill IIEF 15 to assess sexual dysfunction 3 and 9 months after MI.</p><p></p><p>RESULTS: Erectile dysfunction (ED), orgasmic dysfunction, decreased sexual desire, decreased intercourse satisfaction, decreased overall satisfaction were recognized respectively by: 61.3%, 24.2%, 62.9%, 71%, 54.8% of men 3 months after MI, and 51.6%, 17.7%, 58.1%, 77.4%, 59.7% of men 9 months after MI. Men with ED had higher serum CRP (C Reactive Protein) (5.8 vs. 3.8; p = 0.04) and creatinine (1 vs. 0.9; p = 0.04) levels in the peri-infarction period and higher serum BNP* (47.4 vs. 24.6; p = 0.04) measured 3 months after MI than men without ED. They had also higher serum testosterone levels than men without ED (12.6 vs. 10.6; p = 0.03). Men with decreased sexual desire had significantly lower serum DHEAs (dehydroepiandrosterone sulfate) levels in the peri-infarction period than men with normal sexual desire (191.1 vs. 224.3; p = 0.044). Men with ED 9 months after MI had higher serum CRP levels in the peri-infarction period (7.5 vs. 4.6; p = 0.0371). Men with orgasmic dysfunction had lower left ventricular ejection fraction (EF) measured in the peri-infarction period (0.4 vs. 0.5; p = 0.0318).</p><p></p><p>CONCLUSIONS: 1. Men with ED have higher serum CRP and creatinine levels in the peri-infarction period. 2. DHEAs level has an influence on sexual desire and on erectile function after MI. 3. BNP level in post-MI patients affects erectile function. 4. EF has an impact on orgasmic function.</p><p></p><p>Written by: </p><p>Puchalski B, Szyma&#324;ski F, Kowalik R, Filipiak KJ. </p><p></p><p>Reference: Psychiatr Pol. 2013 Sep-Oct;47(5):811-26.</p><p></p><p></p><p>* Brain natriuretic peptide (BNP), now known as B-type natriuretic peptide or Ventricular Natriuretic Peptide (still BNP), is a 32-amino acid polypeptide secreted by the ventricles of the heart in response to excessive stretching of heart muscle cells (cardiomyocytes).</p></blockquote><p></p>
[QUOTE="Nelson Vergel, post: 10596, member: 3"] OBJECTIVES: The aim of the study is to assess the frequency of sexual dysfunction in men after myocardial infarction (MI). METHODS: 62 men were asked to fill IIEF 15 to assess sexual dysfunction 3 and 9 months after MI. RESULTS: Erectile dysfunction (ED), orgasmic dysfunction, decreased sexual desire, decreased intercourse satisfaction, decreased overall satisfaction were recognized respectively by: 61.3%, 24.2%, 62.9%, 71%, 54.8% of men 3 months after MI, and 51.6%, 17.7%, 58.1%, 77.4%, 59.7% of men 9 months after MI. Men with ED had higher serum CRP (C Reactive Protein) (5.8 vs. 3.8; p = 0.04) and creatinine (1 vs. 0.9; p = 0.04) levels in the peri-infarction period and higher serum BNP* (47.4 vs. 24.6; p = 0.04) measured 3 months after MI than men without ED. They had also higher serum testosterone levels than men without ED (12.6 vs. 10.6; p = 0.03). Men with decreased sexual desire had significantly lower serum DHEAs (dehydroepiandrosterone sulfate) levels in the peri-infarction period than men with normal sexual desire (191.1 vs. 224.3; p = 0.044). Men with ED 9 months after MI had higher serum CRP levels in the peri-infarction period (7.5 vs. 4.6; p = 0.0371). Men with orgasmic dysfunction had lower left ventricular ejection fraction (EF) measured in the peri-infarction period (0.4 vs. 0.5; p = 0.0318). CONCLUSIONS: 1. Men with ED have higher serum CRP and creatinine levels in the peri-infarction period. 2. DHEAs level has an influence on sexual desire and on erectile function after MI. 3. BNP level in post-MI patients affects erectile function. 4. EF has an impact on orgasmic function. Written by: Puchalski B, Szymański F, Kowalik R, Filipiak KJ. Reference: Psychiatr Pol. 2013 Sep-Oct;47(5):811-26. * Brain natriuretic peptide (BNP), now known as B-type natriuretic peptide or Ventricular Natriuretic Peptide (still BNP), is a 32-amino acid polypeptide secreted by the ventricles of the heart in response to excessive stretching of heart muscle cells (cardiomyocytes). [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
When Testosterone Is Not Enough
Sexual Dysfunction in Men After a Heart Attack
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