Med student here. I have been on TRT since 21. Here is what I have learned about ED, libido and hormones.

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Hormetheus

Member
What I take for TRT.
I am a med student in my last year. For years I have been on TRT. Because there is just so much misinformation around, I wrote some stuff about what I have learned along the way.

What I found works best for me:

  • 2x 50mg Test cyp per week (s.c. with 30G insulin syringe)
  • 2x 250iu HcG (for fertility as well as steroidogenesis in the adrenals: DHEAS, preg, prog, ect.)
  • I experimented a lot with different doses of aromatase inhibitors, but in the end decided to not use any, because I like the effects a slightly higher estradiol has on my emotionality/personality


Libido issues are NOT the same as erectile dysfunction. Erectile dysfunction can be due to low libido (your brain is not sending signals), nerve damage (your nerves can´t conduct these signals), blood vessel dysfunction (your blood vessels can´t respond to the nerve impusles your brain has sent). In younger males, blood vessels and nerve signals work fine in most cases and ED is due to libido problems, usually because of hormonal problems. In older males ED is roughly about 50% due to libido issues and 50% due to due to blood vessel dysfunction (esp. atherosclerosis: The penis is the antenna of the heart, as one of my professor used to say), or a combination of the two.

Libido depends on a very complex interplay of multiple hormones and neurotransmitter systems. For adequate/good libido (incl. erectile function), multiple hormones need to be adequate. If just a single one of these is “off”, this will be our dealbreaker.

In general, the two most important neurotransmitters when it comes to libido is the delicate balance between dopamine and serotonin signalling. For example, dopaminergic drugs such as selegiline, amphetamine, pramipexole, ect. all have an increased libido as a “side effect”. On the other hand, serotonergic drugs (e.g. SSRIs) will reduce libido.

Multiple hormones affect these neurotransmitter systems.

  • Thyroid hormones: U-shaped curve. Both, very low and very high levels of thyroid hormones lead to libido issues.
  • Cortisol: U-shaped curve. Cortisol pretty much enhances every aspect of dopamine signaling. It increases both the number of dopamine receptors as well as how sensitive the responding cells are to the activation of these receptors.
  • Testosterone: The higher the better. Among other things, testosterone has very powerful effects on dopamine signalling. It also acts on many brain areas important for libido independent of dopamine (e.g. OFC, amygdala, various areas in the hypothalamus, cingulate cortices, insula).
  • DHT: The higher the better. Similar to testosterone it increases dopamine as well as acts on multiple other neural subpopulations important for libido regulation. What is more, DHT increases fluid secretion by the prostate gland. As the prostate gland becomes fuller, libido increases (more to that below).
  • Prolactin: high prolactin inhibits GnRH as well as libido directly.
  • Estradiol: U-shaped curve. Both very low levels and very high levels will reduce libido drastically -in fact crush it to zero. It is no coincidence that one of the major regulators of the overall serotonergic tone is in fact, estradiol.
Note: The factor that most males are most sensitive to is not a change in testosterone levels, but actually a change in estradiol levels. Even small fluctuations can have powerful effects on libido. In my opinion/experience, it is the most common problem when it comes to libido issues (given testosterone levels are not rock bottom). It is also the reason why for many males, libido improves at first after starting TRT, but over time decreases again to low levels. Next to counterregulation in dopaminergic signalling, the reason is, that because levels of estradiol increase over the first weeks of treatment as aromatase expression changes. And as levels of estradiol pass a certain threshold, libido will take a hit. Because of this, adding more testosterone often does make matters worse. In these cases, titration of the RIGHT dosage of an aromatase inhibitor is needed, but unfortunately aromatase inhibitors are highly potent molecules and just a little too much, will crush estrogen levels before any improvements in libido can be noted. Having an experienced doctor helps.


When it comes to hormones, we are all different. Some people don´t notice much change in libido, whether high or low in testosterone or estradiol, whereas for many others a change in these hormones drastically alters libido.

Furthermore, libido increases as levels of oxytocin do (although the main regulator for oxytocin expression is estradiol). a-MSH is important as well, but this is beyond the scope of this article.

One very neglected point is that libido also depends a lot on habit and psychology. Some people will have high libido even with rock bottom levels of testosterone, others will have low libido even if all hormones are optimal. My dad for example had pretty high libido, despite having had levels of free testosterone half of the lower cut-off on the medical reference range. After putting him on TRT, his libido didn´t change much. Similarly, after being castrated some males often maintain their libido for many months to years.

Myth: No fap → rise in GnRH → rise in testosterone → increased libido.The correct order of sequence: No fap → prostate fluid is not emptied anymore → afferent nerve signals recognize the filling of the prostate gland → signal transmission to libido pathways in the brain which sense that the prostate is full and ready for emptying → libido increases → GnRH pulses increase secondarily → small rise in testosterone (but which is too small to affect libido significantly).
Solving libido issues can often be very hard because it depends on soo many factors. If just one of these factors is off, so will libido be. An experienced doctor helps, but unfortunately they are very rare. Most doctors don´t understand anything about hormones, libido regulation, psychology. They just give you Viagra/Cialis, but if your hormones are off, it is not a PDE5i what you need.

I am a medical student in my last year. I started to replace all of my major hormones starting from the age of 23. I wrote a couple of articles to share some of what I have learned, because there is just so much misinformation out there.

Note: This section about libido is part of my guide about how to replace male sex hormones (click here). In it I´ll talk in more detail about all of these things and what to do about it. Had I known then what I know now, it would have saved me lots of time, money, happiness, effort, researching and experimenting. And suffering. I hope you find value in it.
 
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madman

Super Moderator
What I take for TRT.
I am a med student in my last year. For years I have been on TRT. Because there is just so much misinformation around, I wrote some stuff about what I have learned along the way.

What I found works best for me:

  • 2x 50mg Test cyp per week (s.c. with 30G insulin syringe)
  • 2x 250iu HcG (for fertility as well as steroidogenesis in the adrenals: DHEAS, preg, prog, ect.)
  • I experimented a lot with different doses of aromatase inhibitors, but in the end decided to not use any, because I like the effects a slightly higher estradiol has on my emotionality/personality


Libido issues are NOT the same as erectile dysfunction. Erectile dysfunction can be due to low libido (your brain is not sending signals), nerve damage (your nerves can´t conduct these signals), blood vessel dysfunction (your blood vessels can´t respond to the nerve impusles your brain has sent). In younger males, blood vessels and nerve signals work fine in most cases and ED is due to libido problems, usually because of hormonal problems. In older males ED is roughly about 50% due to libido issues and 50% due to due to blood vessel dysfunction (esp. atherosclerosis: The penis is the antenna of the heart, as one of my professor used to say), or a combination of the two.

Libido depends on a very complex interplay of multiple hormones and neurotransmitter systems. For adequate/good libido (incl. erectile function), multiple hormones need to be adequate. If just a single one of these is “off”, this will be our dealbreaker.

In general, the two most important neurotransmitters when it comes to libido is the delicate balance between dopamine and serotonin signalling. For example, dopaminergic drugs such as selegiline, amphetamine, pramipexole, ect. all have an increased libido as a “side effect”. On the other hand, serotonergic drugs (e.g. SSRIs) will reduce libido.

Multiple hormones affect these neurotransmitter systems.

  • Thyroid hormones: U-shaped curve. Both, very low and very high levels of thyroid hormones lead to libido issues.
  • Cortisol: U-shaped curve. Cortisol pretty much enhances every aspect of dopamine signaling. It increases both the number of dopamine receptors as well as how sensitive the responding cells are to the activation of these receptors.
  • Testosterone: The higher the better. Among other things, testosterone has very powerful effects on dopamine signalling. It also acts on many brain areas important for libido independent of dopamine (e.g. OFC, amygdala, various areas in the hypothalamus, cingulate cortices, insula).
  • DHT: The higher the better. Similar to testosterone it increases dopamine as well as acts on multiple other neural subpopulations important for libido regulation. What is more, DHT increases fluid secretion by the prostate gland. As the prostate gland becomes fuller, libido increases (more to that below).
  • Prolactin: high prolactin inhibits GnRH as well as libido directly.
  • Estradiol: U-shaped curve. Both very low levels and very high levels will reduce libido drastically -in fact crush it to zero. It is no coincidence that one of the major regulators of the overall serotonergic tone is in fact, estradiol.
Note: The factor that most males are most sensitive to is not a change in testosterone levels, but actually a change in estradiol levels. Even small fluctuations can have powerful effects on libido. In my opinion/experience, it is the most common problem when it comes to libido issues (given testosterone levels are not rock bottom). It is also the reason why for many males, libido improves at first after starting TRT, but over time decreases again to low levels. Next to counterregulation in dopaminergic signalling, the reason is, that because levels of estradiol increase over the first weeks of treatment as aromatase expression changes. And as levels of estradiol pass a certain threshold, libido will take a hit. Because of this, adding more testosterone often does make matters worse. In these cases, titration of the RIGHT dosage of an aromatase inhibitor is needed, but unfortunately aromatase inhibitors are highly potent molecules and just a little too much, will crush estrogen levels before any improvements in libido can be noted. Having an experienced doctor helps.


When it comes to hormones, we are all different. Some people don´t notice much change in libido, whether high or low in testosterone or estradiol, whereas for many others a change in these hormones drastically alters libido.

Furthermore, libido increases as levels of oxytocin do (although the main regulator for oxytocin expression is estradiol). a-MSH is important as well, but this is beyond the scope of this article.

One very neglected point is that libido also depends a lot on habit and psychology. Some people will have high libido even with rock bottom levels of testosterone, others will have low libido even if all hormones are optimal. My dad for example had pretty high libido, despite having had levels of free testosterone half of the lower cut-off on the medical reference range. After putting him on TRT, his libido didn´t change much. Similarly, after being castrated some males often maintain their libido for many months to years.


Solving libido issues can often be very hard because it depends on soo many factors. If just one of these factors is off, so will libido be. An experienced doctor helps, but unfortunately they are very rare. Most doctors don´t understand anything about hormones, libido regulation, psychology. They just give you Viagra/Cialis, but if your hormones are off, it is not a PDE5i what you need.

I am a medical student in my last year. I started to replace all of my major hormones starting from the age of 23. I wrote a couple of articles to share some of what I have learned, because there is just so much misinformation out there.

Note: This section about libido is part of my guide about how to replace male sex hormones (click here). In it I´ll talk in more detail about all of these things and what to do about it. Had I known then what I know now, it would have saved me lots of time, money, happiness, effort, researching and experimenting. And suffering. I hope you find value in it.

You may want to rethink this statement: In younger males, blood vessels and nerve signals work fine in most cases and ED is due to libido problems, usually because of hormonal problems. In older males ED is roughly about 50% due to libido issues and 50% due to due to blood vessel dysfunction



*This suggests that the aging-related process that leads to ED begins early in life. It turns out that the most common cause of ED, regardless of the patient’s age, is due to a problem with the vascular system of the penis. However, this specific aging-related vascular problem is not caused by arterial disease but due to dysfunction and/or loss of the corporal smooth muscle cells (SMC), the main constituent of the corporal sinusoids.





Down the rabbit hole, we go!








post#4

Background Erectile dysfunction (ED), the most common sexual dysfunction in men, is defined as the inability to achieve or maintain an erection adequate for intercourse (Yafi et al., 2016). ED is caused by psychogenic and organic factors. Organic causes explain up to 80% of ED cases. Organic ED encompasses neurogenic, endocrinologic, vasculogenic, and medication or substance-induced factors. Vasculogenic ED is mostly caused by arterial or inflow disorders, rarely by venous outflow disorders, and is the most prevalent among all cases of organic ED (Ende, 1990).
 

M.J

Well-Known Member
And we still have the million dollar question about how to increase libido yet to be solved.
the only window we get of high libido is when changing protocols.
 

Vince

Super Moderator
What I take for TRT.
I am a med student in my last year. For years I have been on TRT. Because there is just so much misinformation around, I wrote some stuff about what I have learned along the way.

What I found works best for me:

  • 2x 50mg Test cyp per week (s.c. with 30G insulin syringe)
  • 2x 250iu HcG (for fertility as well as steroidogenesis in the adrenals: DHEAS, preg, prog, ect.)
  • I experimented a lot with different doses of aromatase inhibitors, but in the end decided to not use any, because I like the effects a slightly higher estradiol has on my emotionality/personality


Libido issues are NOT the same as erectile dysfunction. Erectile dysfunction can be due to low libido (your brain is not sending signals), nerve damage (your nerves can´t conduct these signals), blood vessel dysfunction (your blood vessels can´t respond to the nerve impusles your brain has sent). In younger males, blood vessels and nerve signals work fine in most cases and ED is due to libido problems, usually because of hormonal problems. In older males ED is roughly about 50% due to libido issues and 50% due to due to blood vessel dysfunction (esp. atherosclerosis: The penis is the antenna of the heart, as one of my professor used to say), or a combination of the two.

Libido depends on a very complex interplay of multiple hormones and neurotransmitter systems. For adequate/good libido (incl. erectile function), multiple hormones need to be adequate. If just a single one of these is “off”, this will be our dealbreaker.

In general, the two most important neurotransmitters when it comes to libido is the delicate balance between dopamine and serotonin signalling. For example, dopaminergic drugs such as selegiline, amphetamine, pramipexole, ect. all have an increased libido as a “side effect”. On the other hand, serotonergic drugs (e.g. SSRIs) will reduce libido.

Multiple hormones affect these neurotransmitter systems.

  • Thyroid hormones: U-shaped curve. Both, very low and very high levels of thyroid hormones lead to libido issues.
  • Cortisol: U-shaped curve. Cortisol pretty much enhances every aspect of dopamine signaling. It increases both the number of dopamine receptors as well as how sensitive the responding cells are to the activation of these receptors.
  • Testosterone: The higher the better. Among other things, testosterone has very powerful effects on dopamine signalling. It also acts on many brain areas important for libido independent of dopamine (e.g. OFC, amygdala, various areas in the hypothalamus, cingulate cortices, insula).
  • DHT: The higher the better. Similar to testosterone it increases dopamine as well as acts on multiple other neural subpopulations important for libido regulation. What is more, DHT increases fluid secretion by the prostate gland. As the prostate gland becomes fuller, libido increases (more to that below).
  • Prolactin: high prolactin inhibits GnRH as well as libido directly.
  • Estradiol: U-shaped curve. Both very low levels and very high levels will reduce libido drastically -in fact crush it to zero. It is no coincidence that one of the major regulators of the overall serotonergic tone is in fact, estradiol.
Note: The factor that most males are most sensitive to is not a change in testosterone levels, but actually a change in estradiol levels. Even small fluctuations can have powerful effects on libido. In my opinion/experience, it is the most common problem when it comes to libido issues (given testosterone levels are not rock bottom). It is also the reason why for many males, libido improves at first after starting TRT, but over time decreases again to low levels. Next to counterregulation in dopaminergic signalling, the reason is, that because levels of estradiol increase over the first weeks of treatment as aromatase expression changes. And as levels of estradiol pass a certain threshold, libido will take a hit. Because of this, adding more testosterone often does make matters worse. In these cases, titration of the RIGHT dosage of an aromatase inhibitor is needed, but unfortunately aromatase inhibitors are highly potent molecules and just a little too much, will crush estrogen levels before any improvements in libido can be noted. Having an experienced doctor helps.


When it comes to hormones, we are all different. Some people don´t notice much change in libido, whether high or low in testosterone or estradiol, whereas for many others a change in these hormones drastically alters libido.

Furthermore, libido increases as levels of oxytocin do (although the main regulator for oxytocin expression is estradiol). a-MSH is important as well, but this is beyond the scope of this article.

One very neglected point is that libido also depends a lot on habit and psychology. Some people will have high libido even with rock bottom levels of testosterone, others will have low libido even if all hormones are optimal. My dad for example had pretty high libido, despite having had levels of free testosterone half of the lower cut-off on the medical reference range. After putting him on TRT, his libido didn´t change much. Similarly, after being castrated some males often maintain their libido for many months to years.


Solving libido issues can often be very hard because it depends on soo many factors. If just one of these factors is off, so will libido be. An experienced doctor helps, but unfortunately they are very rare. Most doctors don´t understand anything about hormones, libido regulation, psychology. They just give you Viagra/Cialis, but if your hormones are off, it is not a PDE5i what you need.

I am a medical student in my last year. I started to replace all of my major hormones starting from the age of 23. I wrote a couple of articles to share some of what I have learned, because there is just so much misinformation out there.

Note: This section about libido is part of my guide about how to replace male sex hormones (click here). In it I´ll talk in more detail about all of these things and what to do about it. Had I known then what I know now, it would have saved me lots of time, money, happiness, effort, researching and experimenting. And suffering. I hope you find value in it.
With that small amount of Hcg, are you still able to remain fertile? Have you had your swimmers checked?

When it comes to estradiol levels, I’ve actually never felt a difference in high or low levels. That could be just me though.
 

Cataceous

Super Moderator
And we still have the million dollar question about how to increase libido yet to be solved.
the only window we get of high libido is when changing protocols.
For me it's been as simple—or as complex—as restoring the suppressed upstream hormones. Previously I had the standard complaint: low libido except when shaking up the protocol. But now I've gone most of the year with normalized libido, long enough to know it's more than a short-term effect.
 

M.J

Well-Known Member
For me it's been as simple—or as complex—as restoring the suppressed upstream hormones. Previously I had the standard complaint: low libido except when shaking up the protocol. But now I've gone most of the year with normalized libido, long enough to know it's more than a short-term effect.
Can you explain more what do you mean normalized libido, during honeymoon periods not only libido is good even morning wood is amazingly great. You don’t need anything to get a good errcrion, but when honey mood period goes away everything back to normal. Or I would say less than normal.
 

Cataceous

Super Moderator
Can you explain more what do you mean normalized libido, during honeymoon periods not only libido is good even morning wood is amazingly great. You don’t need anything to get a good errcrion, but when honey mood period goes away everything back to normal. Or I would say less than normal.
I'm referring to libido that is more raw and visceral, not simply based on habituation. I've seen it described in terms of the reaction to seeing a beautiful women: Do you experience a primal urge or do you feel as though you're simply admiring a nice piece of art? There's also restoration of that anticipatory excitement about sexual activity that was largely absent before. Consistent erections, nocturnal and otherwise, are part of the result.
 

M.J

Well-Known Member
I'm referring to libido that is more raw and visceral, not simply based on habituation. I've seen it described in terms of the reaction to seeing a beautiful women: Do you experience a primal urge or do you feel as though you're simply admiring a nice piece of art? There's also restoration of that anticipatory excitement about sexual activity that was largely absent before. Consistent erections, nocturnal and otherwise, are part of the result.
I don’t mind admiring or getting the urge. My issue is this honey moon period is the only time I really feel like having sex otherwise nothing is happening and as a result in one year of using trt I have yet couldn’t get my wife pregnant and most sex get interrupted by Ed issues or low libido. Yet I am still trying
 

Cataceous

Super Moderator
I don’t mind admiring or getting the urge. My issue is this honey moon period is the only time I really feel like having sex otherwise nothing is happening and as a result in one year of using trt I have yet couldn’t get my wife pregnant and most sex get interrupted by Ed issues or low libido. Yet I am still trying
Yes, I've also found that lack of desire and lack of sensitivity easily leads to ED. On the flip side, I've had honeymoon periods in which libido was downright distracting. Though very high libido is better than low, I think where I am now is preferable; the desire is elicited in the typical ways, such as through visual stimulation, sexual ideation, or even physical contact that's initially non-sexual.

The question that remains is, what fraction of men with reduced libido would respond well to this kind of "turbocharged" HRT? A guy has to be highly motivated to even attempt something like this, so I doubt we'll get an answer anytime soon.
 

Hormetheus

Member
With that small amount of Hcg, are you still able to remain fertile? Have you had your swimmers checked?

When it comes to estradiol levels, I’ve actually never felt a difference in high or low levels. That could be just me though.
Yes. Spermiogram is ok. Could be better. But whenever I need better swimmers I´ll increase the HcG. I also take 2x 50iu FSH per week though
 

JoeMatts

New Member
For me it's been as simple—or as complex—as restoring the suppressed upstream hormones. Previously I had the standard complaint: low libido except when shaking up the protocol. But now I've gone most of the year with normalized libido, long enough to know it's more than a short-term effect.
Apologies if you specified elsewhere, but which particular intervention do you feel made the biggest difference? I've seen you're taking / have taken GnRH, progesterone, hcg, clomid, test p/cyp blend and a few other things, so I'd be curious to hear which one was most impactful.
 

Cataceous

Super Moderator
Apologies if you specified elsewhere, but which particular intervention do you feel made the biggest difference? I've seen you're taking / have taken GnRH, progesterone, hcg, clomid, test p/cyp blend and a few other things, so I'd be curious to hear which one was most impactful.
There could be a synergy involved in getting all these hormones closer to normal levels. But the most obvious positive result came from the GnRH, which with the help of enclomiphene raised LH and FSH to low-normal levels. That was when I felt the biggest improvement in libido. Kisspeptin seems to substantially improve nocturnal erections, with any other effects too subtle to identify.
 

goolapsh

Active Member
I don’t mind admiring or getting the urge. My issue is this honey moon period is the only time I really feel like having sex otherwise nothing is happening and as a result in one year of using trt I have yet couldn’t get my wife pregnant and most sex get interrupted by Ed issues or low libido. Yet I am still trying
What’s your protocol MJ
 

M.J

Well-Known Member
Yes, I've also found that lack of desire and lack of sensitivity easily leads to ED. On the flip side, I've had honeymoon periods in which libido was downright distracting. Though very high libido is better than low, I think where I am now is preferable; the desire is elicited in the typical ways, such as through visual stimulation, sexual ideation, or even physical contact that's initially non-sexual.

The question that remains is, what fraction of men with reduced libido would respond well to this kind of "turbocharged" HRT? A guy has to be highly motivated to even attempt something like this, so I doubt we'll get an answer anytime soon.
One more thing to say here which makes my current libido in my opinion not normal.
I don’t get morning wood. Originally I get that, and also I get it during honey moon period
 

UCFguy01

Active Member
In general, the two most important neurotransmitters when it comes to libido is the delicate balance between dopamine and serotonin signalling. For example, dopaminergic drugs such as selegiline, amphetamine, pramipexole, ect. all have an increased libido as a “side effect”. On the other hand, serotonergic drugs (e.g. SSRIs) will reduce libido.
I find this dopamine serotonin interesting. So what would be an experiment with getting your dopamine up to see if it helps libido? Is there something besides prescription drugs that would help? I'd like to try and increase dopamine to see if it works. I mean, it makes sense to me that it would work.

Would the first honeymoon phase people talk about be because they're dopamine is initially increased at the start of TRT?
 

Jucaro

Active Member
In my particular case, I have found that having very steady levels of testosterone doesn't help on keeping good libido. I have tried several protocols over 4 years on TRT, and it is true that there is a honey moon period when changing protocol, but I have failed to keep a sweet spot at any dose when the protocol provide very steady levels like having T cypionate eod, or even twice a week. I was also on Testosterone undecanoate IM every 2 weeks (average dose 100 mg/week) which makes highly stable levels; the honey moon lasted more, but also faded. I solved the situation decreasing T Undeecanoate to average dose 75 mg /week and complimenting with 25 mg of T Cypionate once a week to make some fluctuation. With that protocol there was no estrogen issues a libido was really good most of the time (but expensive).
Conclusion: Better for me to have slightly fluctuating levels over the week, like my current protocol T cypionate 100 mg once a week. When I use 80 to 100 mg once a week (on Sunday morning) maybe I don´t feel horny by Wednesday but from Thursday to Saturday libido comes very strong, to start fading on Sunday, I take new dose, then again little more libido that night and start fading to repeat the cycle. I feel very comfortable that way. If I add 2,5 mg of daily tadalafilo then I am ready for sex all the time and libido keep good all the time, being the most by Friday to Saturday night (just when most needed…). Tadalafilo in addition to improve my erections, increase greatly my libido when used along with TRT, and I have found it helps me to prevent (to certain levels) high estrogen issues. Problem with Tadalafilo: Makes ejaculation harder to achieve, especially when having sex very frequently.
I also use 250 IU of HCG (Ovitrelle) twice a week. No AI.
 

Hormetheus

Member
I find this dopamine serotonin interesting. So what would be an experiment with getting your dopamine up to see if it helps libido? Is there something besides prescription drugs that would help? I'd like to try and increase dopamine to see if it works. I mean, it makes sense to me that it would work.

Would the first honeymoon phase people talk about be because they're dopamine is initially increased at the start of TRT?

In my guide I do talk about the honeymoon period. Yes, tranisent dopamine upregulation.

No, nothing that you could do without prescription drugs or hormones -at least not something that has any real meaningful effect other than a perhaps slight gain in the single % range.
 

M.J

Well-Known Member
I find this dopamine serotonin interesting. So what would be an experiment with getting your dopamine up to see if it helps libido? Is there something besides prescription drugs that would help? I'd like to try and increase dopamine to see if it works. I mean, it makes sense to me that it would work.

Would the first honeymoon phase people talk about be because they're dopamine is initially increased at the start of TRT?
I got honey moon sometimes when I start or near 40 days and sometimes at the start and at 40 days.
 
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